(Q39841585)

15 September 2017

title

Reactive oxygen species and lipoxygenases regulate the oncogenicity of NPM-ALK-positive anaplastic large cell lymphomas (English)

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15 September 2017

main subject

reactive oxygen species

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K Thornber

1

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15 September 2017

A Colomba

2

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15 September 2017

L Ceccato

3

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15 September 2017

G Delsol

4

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15 September 2017

B Payrastre

5

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15 September 2017

F Gaits-Iacovoni

6

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15 September 2017

language of work or name

English

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publication date

8 June 2009

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15 September 2017

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15 September 2017

volume

28

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15 September 2017

issue

29

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15 September 2017

page(s)

2690-2696

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https://scigraph.springernature.com/pub.10.1038/onc.2009.125

15 September 2017

exact match

0 references

cites work

p130Cas mediates the transforming properties of the anaplastic lymphoma kinase

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.125

Pathobiology of ALK+ anaplastic large-cell lymphoma

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.125

The anaplastic lymphoma kinase in the pathogenesis of cancer

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.125

Hodgkin Reed-Sternberg cells express 15-lipoxygenase-1 and are putative producers of eoxins in vivo: novel insight into the inflammatory features of classical Hodgkin lymphoma.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.125

Activation of Rac1 and the exchange factor Vav3 are involved in NPM-ALK signaling in anaplastic large cell lymphomas.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.125

ROS as signalling molecules: mechanisms that generate specificity in ROS homeostasis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.125

Cyclic AMP-mediated inhibition of 5-lipoxygenase translocation and leukotriene biosynthesis in human neutrophils.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.125

What are cyclooxygenases and lipoxygenases doing in the driver's seat of carcinogenesis?

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.125

SHP1 tyrosine phosphatase negatively regulates NPM-ALK tyrosine kinase signaling

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.125

BCR/ABL kinase induces self-mutagenesis via reactive oxygen species to encode imatinib resistance

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.125

Radical causes of cancer.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.125

Lipoxygenase metabolism: roles in tumor progression and survival

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.125

5-Lipoxygenase: regulation of expression and enzyme activity

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.125

Selective inhibition of arachidonate 5-lipoxygenase by novel acetohydroxamic acids: biochemical assessment in vitro and ex vivo

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.125

5-LOX, 12-LOX and 15-LOX in immature forms of human leukemic blasts.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.125

The signaling mechanism of ROS in tumor progression

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.125

The Tyrosine Phosphatase Shp2 Interacts with NPM-ALK and Regulates Anaplastic Lymphoma Cell Growth and Migration

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.125

Targeting ROS: selective killing of cancer cells by a cruciferous vegetable derived pro-oxidant compound

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.125

31 March 2020

Identifiers

DOI

10.1038/ONC.2009.125

1 reference

15 September 2017

1 reference