(Q39847891)

15 September 2017

title

Menin expression modulates mesenchymal cell commitment to the myogenic and osteogenic lineages (English)

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15 September 2017

Arif Aziz

1

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15 September 2017

Tetsuaki Miyake

2

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15 September 2017

Kurt A Engleka

3

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15 September 2017

Jonathan A Epstein

4

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15 September 2017

John C McDermott

5

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15 September 2017

language of work or name

English

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publication date

21 May 2009

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15 September 2017

published in

Developmental Biology

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15 September 2017

volume

332

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15 September 2017

issue

1

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15 September 2017

page(s)

116-130

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Molecular pathology of the MEN1 gene

15 September 2017

cites work

1 reference

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A mouse model of multiple endocrine neoplasia, type 1, develops multiple endocrine tumors

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Menin is required in cranial neural crest for palatogenesis and perinatal viability

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Menin and TGF-beta superfamily member signaling via the Smad pathway in pituitary, parathyroid and osteoblast

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Mouse embryo fibroblasts lacking the tumor suppressor menin show altered expression of extracellular matrix protein genes.

7 January 2021

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7 January 2021

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7 January 2021

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Menin regulates pancreatic islet growth by promoting histone methylation and expression of genes encoding p27Kip1 and p18INK4c

7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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The muscle regulatory factors MyoD and myf-5 undergo distinct cell cycle-specific expression in muscle cells.

7 January 2021

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Smad7 promotes and enhances skeletal muscle differentiation

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Targeted disruption of Cbfa1 results in a complete lack of bone formation owing to maturational arrest of osteoblasts

7 January 2021

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Activin inhibits pituitary prolactin expression and cell growth through Smads, Pit-1 and menin

7 January 2021

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Regulatory mechanisms that coordinate skeletal muscle differentiation and cell cycle withdrawal.

7 January 2021

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Runx2 is a common target of transforming growth factor beta1 and bone morphogenetic protein 2, and cooperation between Runx2 and Smad5 induces osteoblast-specific gene expression in the pluripotent mesenchymal precursor cell line C2C12.

7 January 2021

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TGF-beta inhibits muscle differentiation through functional repression of myogenic transcription factors by Smad3

7 January 2021

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Phosphorylation of the menin tumor suppressor protein on serine 543 and serine 583

7 January 2021

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Transcriptional control by the TGF-beta/Smad signaling system

7 January 2021

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Type beta transforming growth factor is an inhibitor of myogenic differentiation

7 January 2021

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https://api.crossref.org/works/10.1016%2FJ.YDBIO.2009.05.555

Menin and MLL cooperatively regulate expression of cyclin-dependent kinase inhibitors

7 January 2021

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The novel zinc finger-containing transcription factor osterix is required for osteoblast differentiation and bone formation

7 January 2021

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7 January 2021

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Regulation of myogenic differentiation by type beta transforming growth factor

7 January 2021

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Cbfa1, a candidate gene for cleidocranial dysplasia syndrome, is essential for osteoblast differentiation and bone development

7 January 2021

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Ras/MEK/ERK Up-regulation of the fibroblast KCa channel FIK is a common mechanism for basic fibroblast growth factor and transforming growth factor-beta suppression of myogenesis

7 January 2021

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Activated MEK1 binds the nuclear MyoD transcriptional complex to repress transactivation

7 January 2021

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Menin interacting proteins as clues toward the understanding of multiple endocrine neoplasia type 1.

7 January 2021

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Smad proteins function as co-modulators for MEF2 transcriptional regulatory proteins

7 January 2021

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Regulation of myosin expression during myotome formation

7 January 2021

1 reference

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Homozygous loss of menin is well tolerated in liver, a tissue not affected in MEN1.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YDBIO.2009.05.555

Inactivation of menin, the product of the multiple endocrine neoplasia type 1 gene, inhibits the commitment of multipotential mesenchymal stem cells into the osteoblast lineage

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YDBIO.2009.05.555

Menin is required for bone morphogenetic protein 2- and transforming growth factor beta-regulated osteoblastic differentiation through interaction with Smads and Runx2

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YDBIO.2009.05.555

Menin inactivation leads to loss of transforming growth factor beta inhibition of parathyroid cell proliferation and parathyroid hormone secretion

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YDBIO.2009.05.555

Characterization of the mouse Men1 gene and its expression during development

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YDBIO.2009.05.555

TGF beta signals through a heteromeric protein kinase receptor complex

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YDBIO.2009.05.555

Recombinant human bone morphogenetic protein-2 stimulates osteoblastic maturation and inhibits myogenic differentiation in vitro

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YDBIO.2009.05.555

10.1016/J.YDBIO.2009.05.555

7 January 2021

Identifiers

DOI

1 reference

15 September 2017

1 reference