(Q40061524)

English

The latent membrane protein 1 (LMP1) encoded by Epstein-Barr virus induces expression of the putative oncogene Bcl-3 through activation of the nuclear factor-kappaB.

scientific article published on 25 October 2007

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scholarly article

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Europe PubMed Central

PubMed publication ID

17 September 2017

The latent membrane protein 1 (LMP1) encoded by Epstein-Barr virus induces expression of the putative oncogene Bcl-3 through activation of the nuclear factor-kappaB. (English)

1 reference

Europe PubMed Central

PubMed publication ID

17 September 2017

membrane protein

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based on heuristic

inferred from title

Epstein–Barr virus

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author name string

Hiroyuki Nakamura

1

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Europe PubMed Central

PubMed publication ID

17 September 2017

Chihiro Ishii

2

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Europe PubMed Central

PubMed publication ID

17 September 2017

Masakazu Suehiro

3

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Europe PubMed Central

PubMed publication ID

17 September 2017

Akifumi Iguchi

4

1 reference

Europe PubMed Central

PubMed publication ID

17 September 2017

Kazumichi Kuroda

5

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Europe PubMed Central

PubMed publication ID

17 September 2017

Kazufumi Shimizu

6

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Europe PubMed Central

PubMed publication ID

17 September 2017

Norio Shimizu

7

1 reference

Europe PubMed Central

PubMed publication ID

17 September 2017

Ken-Ichi Imadome

8

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Europe PubMed Central

PubMed publication ID

17 September 2017

Misako Yajima

9

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Europe PubMed Central

PubMed publication ID

17 September 2017

Shigeyoshi Fujiwara

10

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Europe PubMed Central

PubMed publication ID

17 September 2017

publication date

25 October 2007

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Europe PubMed Central

PubMed publication ID

17 September 2017

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Europe PubMed Central

PubMed publication ID

17 September 2017

131

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PubMed publication ID

17 September 2017

2

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17 September 2017

170-179

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Europe PubMed Central

PubMed publication ID

17 September 2017

The oncoprotein Bcl-3 directly transactivates through kappa B motifs via association with DNA-binding p50B homodimers

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

NF-kappa B-inducible BCL-3 expression is an autoregulatory loop controlling nuclear p50/NF-kappa B1 residence

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

Selective activation of NF-kappa B subunits in human breast cancer: potential roles for NF-kappa B2/p52 and for Bcl-3

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

Association of TRAF1, TRAF2, and TRAF3 with an Epstein-Barr virus LMP1 domain important for B-lymphocyte transformation: role in NF-kappaB activation

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

Activation of the p38 mitogen-activated protein kinase pathway by Epstein-Barr virus-encoded latent membrane protein 1 coregulates interleukin-6 and interleukin-8 production

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

Activation of the cJun N-terminal kinase (JNK) pathway by the Epstein-Barr virus-encoded latent membrane protein 1 (LMP1).

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

The candidate oncoprotein Bcl-3 is an antagonist of p50/NF-kappa B-mediated inhibition

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https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

The candidate proto-oncogene bcl-3 encodes a transcriptional coactivator that activates through NF-kappa B p50 homodimers

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

Latent membrane protein 1 of Epstein-Barr virus interacts with JAK3 and activates STAT proteins

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

Latent membrane protein 1 of Epstein-Barr virus mimics a constitutively active receptor molecule.

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

Constitutive activation of NF-kappaB causes resistance to apoptosis in human cutaneous T cell lymphoma HuT-78 cells. Autocrine role of tumor necrosis factor and reactive oxygen intermediates

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

Epstein-Barr virus in nasal T-cell lymphomas in patients with lethal midline granuloma

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

Phenotypic modulation of Hodgkin and Reed-Sternberg cells by Epstein-Barr virus

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

The Epstein-Barr virus oncoprotein latent membrane protein 1 engages the tumor necrosis factor receptor-associated proteins TRADD and receptor-interacting protein (RIP) but does not induce apoptosis or require RIP for NF-kappaB activation

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

LMP-1's transmembrane domains encode multiple functions required for LMP-1's efficient signaling

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

Epstein-Barr virus latent membrane protein-1 triggers AP-1 activity via the c-Jun N-terminal kinase cascade

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

Epstein-Barr virus-mediated B-cell proliferation is dependent upon latent membrane protein 1, which simulates an activated CD40 receptor

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

Cyld inhibits tumor cell proliferation by blocking Bcl-3-dependent NF-kappaB signaling

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

Elevated NF-kappaB p50 complex formation and Bcl-3 expression in classical Hodgkin, anaplastic large-cell, and other peripheral T-cell lymphomas

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

Cloning of the chromosome translocation breakpoint junction of the t(14;19) in chronic lymphocytic leukemia

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

Immunological adjuvants promote activated T cell survival via induction of Bcl-3.

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

The Epstein-Barr virus transforming protein LMP1 engages signaling proteins for the tumor necrosis factor receptor family

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

Characterization of novel natural killer (NK)-cell and gammadelta T-cell lines established from primary lesions of nasal T/NK-cell lymphomas associated with the Epstein-Barr virus

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

Global changes in Kaposi's sarcoma-associated virus gene expression patterns following expression of a tetracycline-inducible Rta transactivator

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

The bcl-3 proto-oncogene encodes a nuclear I kappa B-like molecule that preferentially interacts with NF-kappa B p50 and p52 in a phosphorylation-dependent manner

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

The candidate proto-oncogene bcl-3 is related to genes implicated in cell lineage determination and cell cycle control

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

Lymphadenopathy, splenomegaly, and altered immunoglobulin production in BCL3 transgenic mice.

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

Preferential expansion of Vgamma9-JgammaP/Vdelta2-Jdelta3 gammadelta T cells in nasal T-cell lymphoma and chronic active Epstein-Barr virus infection

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

Bcl-3 expression promotes cell survival following interleukin-4 deprivation and is controlled by AP1 and AP1-like transcription factors

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

Tumorigenicity of mouse BALB/c 3T3 fibroblast cells which express Epstein-Barr virus-encoded LMP1 and show normal growth phenotypes in vitro is correlated with loss of transforming growth factor-beta 1-mediated growth inhibition

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

Cutting edge: Bcl-3 up-regulation by signal 3 cytokine (IL-12) prolongs survival of antigen-activated CD8 T cells

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

The putative oncoprotein Bcl-3 induces cyclin D1 to stimulate G(1) transition

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

N- and C-terminal sequences control degradation of MAD3/I kappa B alpha in response to inducers of NF-kappa B activity

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

Candidate proto-oncogene bcl-3 encodes a subunit-specific inhibitor of transcription factor NF-kappa B

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

Heterogeneous, restricted patterns of Epstein-Barr virus (EBV) latent gene expression in patients with chronic active EBV infection

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

Latency pattern of Epstein-Barr virus and methylation status in Epstein-Barr virus-associated hemophagocytic syndrome

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

Common cytological and cytogenetic features of Epstein-Barr virus (EBV)-positive natural killer (NK) cells and cell lines derived from patients with nasal T/NK-cell lymphomas, chronic active EBV infection and hydroa vacciniforme-like eruptions

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

Epstein-Barr virus latent membrane protein (LMP1) is not sufficient to maintain proliferation of B cells but both it and activated CD40 can prolong their survival.

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIRUSRES.2007.09.003

7 January 2021

Identifiers

10.1016/J.VIRUSRES.2007.09.003

1 reference

Europe PubMed Central

PubMed publication ID

17 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

17 September 2017

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