(Q40183990)

18 September 2017

title

Oridonin induced apoptosis through Akt and MAPKs signaling pathways in human osteosarcoma cells (English)

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18 September 2017

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Song Jin

1

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18 September 2017

Jing-Nan Shen

2

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18 September 2017

Jin Wang

3

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18 September 2017

Gang Huang

4

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18 September 2017

Jia-Guo Zhou

5

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18 September 2017

publication date

26 February 2007

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full work available at URL

18 September 2017

https://www.tandfonline.com/doi/pdf/10.4161/cbt.6.2.3621?needAccess=true

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19 March 2024

https://api.unpaywall.org/v2/10.4161/cbt.6.2.3621?email=unpaywall_01@example.com

published in

Cancer Biology and Therapy

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18 September 2017

volume

6

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18 September 2017

issue

2

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18 September 2017

page(s)

261-268

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https://api.unpaywall.org/v2/10.4161/cbt.6.2.3621?email=unpaywall_01@example.com

18 September 2017

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19 March 2024

access restriction status

open access

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based on heuristic

inferred from online access status

cites work

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Cytochrome c release from oridonin-treated apoptotic A375-S2 cells is dependent on p53 and extracellular signal-regulated kinase activation

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Akt phosphorylation of BAD couples survival signals to the cell-intrinsic death machinery

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Akt promotes cell survival by phosphorylating and inhibiting a Forkhead transcription factor

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Oridonin, a diterpenoid purified from Rabdosia rubescens, inhibits the proliferation of cells from lymphoid malignancies in association with blockade of the NF-kappa B signal pathways

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Activation of phosphoinositide 3-kinase, protein kinase C, and extracellular signal-regulated kinase is required for oridonin-enhanced phagocytosis of apoptotic bodies in human macrophage-like U937 cells

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12 April 2024

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12 April 2024

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c-Jun N-terminal kinase/stress-activated protein kinase (JNK/SAPK) is required for mitoxantrone- and anisomycin-induced apoptosis in HL-60 cells

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https://opencitations.net/index/api/v1/references/10.4161/CBT.6.2.3621

Identifiers

DOI

10.4161/CBT.6.2.3621

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18 September 2017

1 reference