(Q40337889)

English

E-cadherin modulates Wnt-dependent transcription in colorectal cancer cells but does not alter Wnt-independent gene expression in fibroblasts

scientific article published on February 2006

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scholarly article

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Europe PubMed Central

PubMed publication ID

19 September 2017

E-cadherin modulates Wnt-dependent transcription in colorectal cancer cells but does not alter Wnt-independent gene expression in fibroblasts (English)

1 reference

Europe PubMed Central

PubMed publication ID

19 September 2017

colorectal cancer

1 reference

based on heuristic

inferred from title

colorectal carcinoma

1 reference

based on heuristic

inferred from title

author name string

Felix Kuphal

1

1 reference

Europe PubMed Central

PubMed publication ID

19 September 2017

Jürgen Behrens

2

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Europe PubMed Central

PubMed publication ID

19 September 2017

language of work or name

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publication date

1 February 2006

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Europe PubMed Central

PubMed publication ID

19 September 2017

Experimental Cell Research

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Europe PubMed Central

PubMed publication ID

19 September 2017

312

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Europe PubMed Central

PubMed publication ID

19 September 2017

4

1 reference

Europe PubMed Central

PubMed publication ID

19 September 2017

457-467

1 reference

Europe PubMed Central

PubMed publication ID

19 September 2017

Sticky business: orchestrating cellular signals at adherens junctions

1 reference

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Alpha-catenin: at the junction of intercellular adhesion and actin dynamics

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E-cadherin null mutant embryos fail to form a trophectoderm epithelium

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A targeted mutation in the mouse E-cadherin gene results in defective preimplantation development

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E-cadherin controls adherens junctions in the epidermis and the renewal of hair follicles

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E-cadherin is a survival factor for the lactating mouse mammary gland

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Cadherins and catenins: role in signal transduction and tumor progression

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E-cadherin-mediated cell-cell adhesion prevents invasiveness of human carcinoma cells

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Genetic manipulation of E-cadherin expression by epithelial tumor cells reveals an invasion suppressor role

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A causal role for E-cadherin in the transition from adenoma to carcinoma

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Epithelial-mesenchymal transitions in tumour progression

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The transcription factor snail is a repressor of E-cadherin gene expression in epithelial tumour cells

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The transcription factor snail controls epithelial-mesenchymal transitions by repressing E-cadherin expression

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Loss of epithelial differentiation and gain of invasiveness correlates with tyrosine phosphorylation of the E-cadherin/beta-catenin complex in cells transformed with a temperature-sensitive v-SRC gene

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Hakai, a c-Cbl-like protein, ubiquitinates and induces endocytosis of the E-cadherin complex

1 reference

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Silencing of the E-cadherin invasion-suppressor gene by CpG methylation in human carcinomas

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Biochemical interactions in the wnt pathway

1 reference

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Functional interaction of beta-catenin with the transcription factor LEF-1

1 reference

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XTcf-3 transcription factor mediates beta-catenin-induced axis formation in Xenopus embryos

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Linking colorectal cancer to Wnt signaling

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Wnt signalling in stem cells and cancer

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Autoregulation of E-cadherin expression by cadherin-cadherin interactions: the roles of beta-catenin signaling, Slug, and MAPK

1 reference

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E-Cadherin Suppresses Cellular Transformation by Inhibiting β-Catenin Signaling in an Adhesion-Independent Manner

1 reference

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Convergence of Wnt, beta-catenin, and cadherin pathways

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Tight control of gene expression in mammalian cells by tetracycline-responsive promoters

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Expressed recombinant cadherins mediate cell sorting in model systems

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Armadillo coactivates transcription driven by the product of the Drosophila segment polarity gene dTCF

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The LIM-only protein FHL2 interacts with beta-catenin and promotes differentiation of mouse myoblasts

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Single-chain Tet transregulators

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Dissecting tumor cell invasion: epithelial cells acquire invasive properties after the loss of uvomorulin-mediated cell-cell adhesion

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Expression of ribosomal and translation-associated genes is correlated with a favorable clinical course in chronic lymphocytic leukemia.

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Transcriptional regulation by the phosphorylation-dependent factor CREB

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Genome-wide analysis of cAMP-response element binding protein occupancy, phosphorylation, and target gene activation in human tissues

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Cadherin-mediated cellular signaling

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Binding to cadherins antagonizes the signaling activity of beta-catenin during axis formation in Xenopus

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The cyclin D1 gene is a target of the beta-catenin/LEF-1 pathway

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Nuclear export of the APC tumour suppressor controls beta-catenin function in transcription

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Variable beta-catenin expression in colorectal cancers indicates tumor progression driven by the tumor environment

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Wnt-dependent regulation of the E-cadherin repressor snail

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Activation of the protein kinase Akt/PKB by the formation of E-cadherin-mediated cell-cell junctions. Evidence for the association of phosphatidylinositol 3-kinase with the E-cadherin adhesion complex.

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Induction of mutual stabilization and retardation of tumor growth by coexpression of plakoglobin and E-cadherin in mouse skin spindle carcinoma cells

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Adaptation of core mechanisms to generate cell polarity

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Rac Activation upon Cell-Cell Contact Formation Is Dependent on Signaling from the Epidermal Growth Factor Receptor

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Identifiers

10.1016/J.YEXCR.2005.11.007

1 reference

Europe PubMed Central

PubMed publication ID

19 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

19 September 2017

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