(Q40399437)

20 September 2017

title

Caspase-9 and effector caspases have sequential and distinct effects on mitochondria (English)

1 reference

20 September 2017

Enrique Cepero

1

1 reference

20 September 2017

Anne M King

2

1 reference

20 September 2017

Lane M Coffey

3

1 reference

20 September 2017

Rebeka G Perez

4

1 reference

20 September 2017

Lawrence H Boise

5

1 reference

20 September 2017

language of work or name

English

0 references

publication date

1 September 2005

1 reference

20 September 2017

1 reference

20 September 2017

volume

24

1 reference

20 September 2017

issue

42

1 reference

20 September 2017

page(s)

6354-6366

1 reference

https://scigraph.springernature.com/pub.10.1038/sj.onc.1208793

20 September 2017

exact match

0 references

cites work

Human xenomitochondrial cybrids. Cellular models of mitochondrial complex I deficiency

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208793

Caspase inhibition extends the commitment to neuronal death beyond cytochrome c release to the point of mitochondrial depolarization

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208793

Inhibition of human caspases by peptide-based and macromolecular inhibitors

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208793

Arachidonic acid converts the glutathione depletion-induced apoptosis to necrosis by promoting lipid peroxidation and reducing caspase-3 activity in rat glioma cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208793

The pro-apoptotic proteins, Bid and Bax, cause a limited permeabilization of the mitochondrial outer membrane that is enhanced by cytosol

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208793

The release of cytochrome c from mitochondria during apoptosis of NGF-deprived sympathetic neurons is a reversible event

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208793

Caspase-mediated loss of mitochondrial function and generation of reactive oxygen species during apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208793

A comparative study of apoptosis and necrosis in HepG2 cells: oxidant-induced caspase inactivation leads to necrosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208793

Proapoptotic BH3-only Bcl-2 family members induce cytochrome c release, but not mitochondrial membrane potential loss, and do not directly modulate voltage-dependent anion channel activity

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208793

Preservation of mitochondrial structure and function after Bid- or Bax-mediated cytochrome c release

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208793

Cytochrome c maintains mitochondrial transmembrane potential and ATP generation after outer mitochondrial membrane permeabilization during the apoptotic process

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208793

Superoxide in apoptosis. Mitochondrial generation triggered by cytochrome c loss

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208793

7 January 2021

Cell death: critical control points

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208793

7 January 2021

BCL-2 family members and the mitochondria in apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208793

7 January 2021

Caspase cleaved BID targets mitochondria and is required for cytochrome c release, while BCL-XL prevents this release but not tumor necrosis factor-R1/Fas death

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208793

7 January 2021

Differential requirement for caspase 9 in apoptotic pathways in vivo.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208793

7 January 2021

Regulation of reactive oxygen species-induced apoptosis and necrosis by caspase 3-like proteases

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208793

7 January 2021

Bcl-xL inhibits cytochrome c release but not mitochondrial depolarization during the activation of multiple death pathways by tumor necrosis factor-alpha

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208793

7 January 2021

Bid-induced cytochrome c release is mediated by a pathway independent of mitochondrial permeability transition pore and Bax.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208793

7 January 2021

The ADP/ATP translocator is not essential for the mitochondrial permeability transition pore

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208793

7 January 2021

Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208793

7 January 2021

Intrinsic tumour suppression

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208793

7 January 2021

The permeability transition pore complex: a target for apoptosis regulation by caspases and bcl-2-related proteins

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208793

7 January 2021

Caspases disrupt mitochondrial membrane barrier function.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208793

7 January 2021

Apoptosis by death factor

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208793

7 January 2021

Disruption of mitochondrial function during apoptosis is mediated by caspase cleavage of the p75 subunit of complex I of the electron transport chain

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208793

7 January 2021

Oxidative phosphorylation at the fin de siècle

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208793

7 January 2021

Bcl-xL regulates the membrane potential and volume homeostasis of mitochondria

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208793

7 January 2021

Proapoptotic BAX and BAK: a requisite gateway to mitochondrial dysfunction and death

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208793

7 January 2021

Apaf1 is required for mitochondrial pathways of apoptosis and brain development

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208793

7 January 2021

Identifiers

DOI

10.1038/SJ.ONC.1208793

1 reference

Dimensions Publication ID

20 September 2017

1005885170

0 references

1 reference