(Q40399600)

20 September 2017

Puma(*)Mcl-1 interaction is not sufficient to prevent rapid degradation of Mcl-1. (English)

1 reference

20 September 2017

Yide Mei

1

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20 September 2017

Wenjing Du

series ordinal

2

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20 September 2017

Yonghui Yang

series ordinal

3

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20 September 2017

Mian Wu

series ordinal

4

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20 September 2017

language of work or name

English

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publication date

1 November 2005

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20 September 2017

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20 September 2017

24

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20 September 2017

7224-7237

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20 September 2017

48

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https://scigraph.springernature.com/pub.10.1038/sj.onc.1208873

20 September 2017

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cites work

Regulation of apoptosis and cell cycle progression by MCL1. Differential role of proliferating cell nuclear antigen

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208873

Expression of bbc3, a pro-apoptotic BH3-only gene, is regulated by diverse cell death and survival signals

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208873

MCL1, a gene expressed in programmed myeloid cell differentiation, has sequence similarity to BCL2

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208873

BCL-2 and MCL-1 expression in Chinese hamster ovary cells inhibits intracellular acidification and apoptosis induced by staurosporine

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208873

Unreliability of the cytochrome c-enhanced green fluorescent fusion protein as a marker of cytochrome c release in cells that overexpress Bcl-2.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208873

Live or let die: the cell's response to p53

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208873

The intracellular distribution and pattern of expression of Mcl-1 overlap with, but are not identical to, those of Bcl-2

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208873

PUMA mediates the apoptotic response to p53 in colorectal cancer cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208873

Thymocyte apoptosis induced by p53-dependent and independent pathways.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208873

7 January 2021

Antisense strategy shows that Mcl-1 rather than Bcl-2 or Bcl-x(L) is an essential survival protein of human myeloma cells.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208873

7 January 2021

Puma is an essential mediator of p53-dependent and -independent apoptotic pathways

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208873

7 January 2021

Substrate targeting in the ubiquitin system

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208873

7 January 2021

Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208873

7 January 2021

p53 is required for radiation-induced apoptosis in mouse thymocytes

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208873

7 January 2021

Development and maintenance of B and T lymphocytes requires antiapoptotic MCL-1.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208873

7 January 2021

Defying death after DNA damage.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208873

7 January 2021

Amino acid sequences common to rapidly degraded proteins: the PEST hypothesis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208873

7 January 2021

Dissecting p53 tumor suppressor functions in vivo

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208873

7 January 2021

Surfing the p53 network

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208873

7 January 2021

MCL-1, a member of the BLC-2 family, is induced rapidly in response to signals for cell differentiation or death, but not to signals for cell proliferation

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208873

7 January 2021

BH1 and BH2 domains of Bcl-2 are required for inhibition of apoptosis and heterodimerization with Bax.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208873

7 January 2021

Induction of BCL2 family member MCL1 as an early response to DNA damage.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208873

7 January 2021

Identifiers

DOI

10.1038/SJ.ONC.1208873

1 reference

Dimensions Publication ID

20 September 2017

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1 reference