(Q40451404)

20 September 2017

Cross-presentation by intercellular peptide transfer through gap junctions. (English)

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20 September 2017

6

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author name string

Joost Neijssen

1

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20 September 2017

Carla Herberts

2

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20 September 2017

Jan Wouter Drijfhout

3

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20 September 2017

Eric Reits

4

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20 September 2017

Lennert Janssen

5

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20 September 2017

language of work or name

English

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publication date

1 March 2005

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20 September 2017

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20 September 2017

434

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20 September 2017

83-88

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20 September 2017

7029

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https://scigraph.springernature.com/pub.10.1038/nature03290

20 September 2017

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cites work

Making sense of mass destruction: quantitating MHC class I antigen presentation

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https://api.crossref.org/works/10.1038%2FNATURE03290

7 January 2021

The ins and outs of cross-presentation

1 reference

https://api.crossref.org/works/10.1038%2FNATURE03290

7 January 2021

Cellular mechanisms governing cross-presentation of exogenous antigens

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https://api.crossref.org/works/10.1038%2FNATURE03290

7 January 2021

Cross-presentation, dendritic cell subsets, and the generation of immunity to cellular antigens

1 reference

https://api.crossref.org/works/10.1038%2FNATURE03290

7 January 2021

Regulation of connexin biosynthesis, assembly, gap junction formation, and removal

1 reference

https://api.crossref.org/works/10.1038%2FNATURE03290

7 January 2021

Selective permeability of gap junction channels

1 reference

https://api.crossref.org/works/10.1038%2FNATURE03290

7 January 2021

Regulation of gap junctions by tyrosine protein kinases

1 reference

https://api.crossref.org/works/10.1038%2FNATURE03290

7 January 2021

Gap junctions and connexin-mediated communication in the immune system

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https://api.crossref.org/works/10.1038%2FNATURE03290

7 January 2021

Dynamics of connexin43 phosphorylation in pp60v-src-transformed cells

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https://api.crossref.org/works/10.1038%2FNATURE03290

7 January 2021

In vitro growth inhibition of neoplastically transformed cells by non-transformed cells: requirement for gap junctional intercellular communication.

1 reference

https://api.crossref.org/works/10.1038%2FNATURE03290

7 January 2021

HSV-2 disrupts gap junctional intercellular communication between mammalian cells in vitro.

1 reference

https://api.crossref.org/works/10.1038%2FNATURE03290

7 January 2021

Human papillomavirus type 16 E5 protein affects cell-cell communication in an epithelial cell line

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https://api.crossref.org/works/10.1038%2FNATURE03290

7 January 2021

Interaction of c-Src with gap junction protein connexin-43. Role in the regulation of cell-cell communication

1 reference

https://api.crossref.org/works/10.1038%2FNATURE03290

7 January 2021

Peptide diffusion, protection, and degradation in nuclear and cytoplasmic compartments before antigen presentation by MHC class I.

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https://api.crossref.org/works/10.1038%2FNATURE03290

7 January 2021

Besides affecting intracellular calcium signaling, 2-APB reversibly blocks gap junctional coupling in confluent monolayers, thereby allowing measurement of single-cell membrane currents in undissociated cells

1 reference

https://api.crossref.org/works/10.1038%2FNATURE03290

7 January 2021

Quantitating protein synthesis, degradation, and endogenous antigen processing

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https://api.crossref.org/works/10.1038%2FNATURE03290

7 January 2021

TNF-alpha plus IFN-gamma induce connexin43 expression and formation of gap junctions between human monocytes/macrophages that enhance physiological responses

1 reference

https://api.crossref.org/works/10.1038%2FNATURE03290

7 January 2021

Presentation of antigen by endothelial cells and chemoattraction are required for homing of insulin-specific CD8+ T cells.

1 reference

https://api.crossref.org/works/10.1038%2FNATURE03290

7 January 2021

Roles of heat-shock proteins in antigen presentation and cross-presentation

1 reference

https://api.crossref.org/works/10.1038%2FNATURE03290

7 January 2021

Recycling MHC class I molecules and endosomal peptide loading

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https://api.crossref.org/works/10.1038%2FNATURE03290

7 January 2021

Control of cross-presentation during dendritic cell maturation

1 reference

https://api.crossref.org/works/10.1038%2FNATURE03290

7 January 2021

ER-phagosome fusion defines an MHC class I cross-presentation compartment in dendritic cells

1 reference

https://api.crossref.org/works/10.1038%2FNATURE03290

7 January 2021

Phagosomes are competent organelles for antigen cross-presentation

1 reference

https://api.crossref.org/works/10.1038%2FNATURE03290

7 January 2021

Cutting edge: efficient MHC class I cross-presentation during early vaccinia infection requires the transfer of proteasomal intermediates between antigen donor and presenting cells

1 reference

https://api.crossref.org/works/10.1038%2FNATURE03290

7 January 2021

Antigen bias in T cell cross-priming

1 reference

https://api.crossref.org/works/10.1038%2FNATURE03290

7 January 2021

CD8+ T cell cross-priming via transfer of proteasome substrates

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https://api.crossref.org/works/10.1038%2FNATURE03290

7 January 2021

Cellular protein is the source of cross-priming antigen in vivo

1 reference

https://api.crossref.org/works/10.1038%2FNATURE03290

7 January 2021

Human telomerase reverse transcriptase-transduced human cytotoxic T cells suppress the growth of human melanoma in immunodeficient mice.

1 reference

https://api.crossref.org/works/10.1038%2FNATURE03290

7 January 2021

Identifiers

DOI

10.1038/NATURE03290

1 reference

Dimensions Publication ID

20 September 2017

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