(Q40463608)

20 September 2017

title

HOXA9 activates transcription of the gene encoding gp91Phox during myeloid differentiation. (English)

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20 September 2017

Ling Bei

1

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20 September 2017

YuFeng Lu

series ordinal

2

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20 September 2017

Elizabeth A Eklund

series ordinal

3

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20 September 2017

language of work or name

English

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publication date

28 January 2005

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Journal of Biological Chemistry

20 September 2017

published in

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20 September 2017

volume

280

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20 September 2017

issue

13

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20 September 2017

page(s)

12359-12370

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The human HOX gene family

20 September 2017

cites work

1 reference

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The Oncogene Nup98-HOXA9 Induces Gene Transcription in Myeloid Cells

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Hoxa9 immortalizes a granulocyte-macrophage colony-stimulating factor-dependent promyelocyte capable of biphenotypic differentiation to neutrophils or macrophages, independent of enforced meis expression

21 January 2018

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Overexpression of the myeloid leukemia-associated Hoxa9 gene in bone marrow cells induces stem cell expansion

21 January 2018

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Hoxa9 transforms primary bone marrow cells through specific collaboration with Meis1a but not Pbx1b

21 January 2018

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Cloning the gene for an inherited human disorder--chronic granulomatous disease--on the basis of its chromosomal location

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Molecular classification of cancer: class discovery and class prediction by gene expression monitoring

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MLL translocations specify a distinct gene expression profile that distinguishes a unique leukemia

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An Mll-AF9 fusion gene made by homologous recombination causes acute leukemia in chimeric mice: a method to create fusion oncogenes

21 January 2018

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The t(7;11)(p15;p15) translocation in acute myeloid leukaemia fuses the genes for nucleoporin NUP98 and class I homeoprotein HOXA9

21 January 2018

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NUP98-HOXA9 expression in hemopoietic stem cells induces chronic and acute myeloid leukemias in mice

21 January 2018

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Pbx modulation of Hox homeodomain amino-terminal arms establishes different DNA-binding specificities across the Hox locus

21 January 2018

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Cell signaling switches HOX-PBX complexes from repressors to activators of transcription mediated by histone deacetylases and histone acetyltransferases.

21 January 2018

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HOXA9 forms triple complexes with PBX2 and MEIS1 in myeloid cells

21 January 2018

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Meis1a suppresses differentiation by G-CSF and promotes proliferation by SCF: potential mechanisms of cooperativity with Hoxa9 in myeloid leukemia

21 January 2018

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Tyrosine phosphorylation of HoxA10 decreases DNA binding and transcriptional repression during interferon gamma -induced differentiation of myeloid leukemia cell lines

21 January 2018

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HoxA10 Represses Gene Transcription in Undifferentiated Myeloid Cells by Interaction with Histone Deacetylase 2

21 January 2018

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SHP1 protein-tyrosine phosphatase regulates HoxA10 DNA binding and transcriptional repression activity in undifferentiated myeloid cells

21 January 2018

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Overexpression of HOXA10 in murine hematopoietic cells perturbs both myeloid and lymphoid differentiation and leads to acute myeloid leukemia

21 January 2018

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Accurate transcription initiation by RNA polymerase II in a soluble extract from isolated mammalian nuclei

21 January 2018

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SHP1 protein-tyrosine phosphatase inhibits gp91PHOX and p67PHOX expression by inhibiting interaction of PU.1, IRF1, interferon consensus sequence-binding protein, and CREB-binding protein with homologous Cis elements in the CYBB and NCF2 genes

21 January 2018

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Identification of unknown target genes of human transcription factors using chromatin immunoprecipitation

21 January 2018

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A model for extradenticle function as a switch that changes HOX proteins from repressors to activators

21 January 2018

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The transcriptome of the leukemogenic homeoprotein HOXA9 in human hematopoietic cells

21 January 2018

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Structure of HoxA9 and Pbx1 bound to DNA: Hox hexapeptide and DNA recognition anterior to posterior

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M408138200

21 January 2018

Identifiers

DOI

10.1074/JBC.M408138200

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20 September 2017

1 reference