(Q40481967)

20 September 2017

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title

The pathogenesis of septic shock. (English)

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20 September 2017

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Sriskandan S

1

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20 September 2017

Cohen J

series ordinal

2

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20 September 2017

publication date

1 May 1995

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20 September 2017

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20 September 2017

volume

30

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20 September 2017

issue

3

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20 September 2017

page(s)

201-206

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Plasma tumor necrosis factor in patients with septic shock. Mortality rate, incidence of adult respiratory distress syndrome, and effects of methylprednisolone administration.

20 September 2017

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7 January 2021

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Detection of interleukin 8 and tumor necrosis factor in normal humans after intravenous endotoxin: the effect of antiinflammatory agents

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T-cell activation by superantigens

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7 January 2021

Cytokine signal transduction

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A receptor for tumor necrosis factor defines an unusual family of cellular and viral proteins

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Signal transduction by lymphocyte antigen receptors

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Lymphokine production by human T cells in disease states

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Tumor necrosis factor in the pathophysiology of infection and sepsis

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Pathogenesis of the toxic shock syndrome: T cell mediated lethal shock caused by the superantigen TSST-1

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Mice deficient for the 55 kd tumor necrosis factor receptor are resistant to endotoxic shock, yet succumb to L. monocytogenes infection

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Decreased sensitivity to tumour-necrosis factor but normal T-cell development in TNF receptor-2-deficient mice

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Plasma cytokine and endotoxin levels correlate with survival in patients with the sepsis syndrome

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Traffic signals for lymphocyte recirculation and leukocyte emigration: the multistep paradigm

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Inhibition of nitric oxide synthase in experimental gram-negative sepsis.

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A circulating myocardial depressant substance in humans with septic shock. Septic shock patients with a reduced ejection fraction have a circulating factor that depresses in vitro myocardial cell performance

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7 January 2021

Mechanisms of myocardial depression in sepsis

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7 January 2021

Administration of anti-TNF antibody improves left ventricular function in septic shock patients. Results of a pilot study

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Tumor necrosis factor-alpha as a myocardial depressant substance

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Cellular basis for the negative inotropic effects of tumor necrosis factor-alpha in the adult mammalian heart

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Nitric oxide synthases and cardiovascular signaling

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Negative inotropic effects of cytokines on the heart mediated by nitric oxide

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7 January 2021

10.1016/S0163-4453(95)90647-9

Identifiers

DOI

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20 September 2017

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