(Q40489599)

21 September 2017

title

Effect of IGF-1 on the balance between autophagy of dysfunctional mitochondria and apoptosis (English)

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21 September 2017

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Yiping Gu

1

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21 September 2017

Chunjie Wang

series ordinal

2

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21 September 2017

Amos Cohen

series ordinal

3

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21 September 2017

language of work or name

English

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publication date

1 November 2004

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21 September 2017

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21 September 2017

volume

577

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21 September 2017

issue

3

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21 September 2017

page(s)

357-360

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Premature ageing in mice expressing defective mitochondrial DNA polymerase

21 September 2017

cites work

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2004.10.040

Molecular Biology of Aging

21 January 2018

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https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2004.10.040

Cell sorting experiments link persistent mitochondrial DNA damage with loss of mitochondrial membrane potential and apoptotic cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2004.10.040

Mitochondrial DNA damage is more extensive and persists longer than nuclear DNA damage in human cells following oxidative stress

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2004.10.040

Autophagy genes are essential for dauer development and life-span extension in C. elegans

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2004.10.040

Healthy Animals with Extreme Longevity

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2004.10.040

Genetics and the specificity of the aging process

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2004.10.040

IGF-1 receptor regulates lifespan and resistance to oxidative stress in mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2004.10.040

Intracellular heteroplasmy for disease-associated point mutations in mtDNA: implications for disease expression and evidence for mitotic segregation of heteroplasmic units of mtDNA.

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2004.10.040

Bcl-2 family proteins and mitochondria.

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2004.10.040

Oxidative stress, caloric restriction, and aging

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2004.10.040

Mitochondria and apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2004.10.040

A structural view of mitochondria-mediated apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2004.10.040

Mitochondrial Genome Evolution and the Origin of Eukaryotes

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2004.10.040

21 January 2018

Mitochondrial evolution

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2004.10.040

Unique mitochondrial genome architecture in unicellular relatives of animals

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2004.10.040

MELAS mutation in mtDNA binding site for transcription termination factor causes defects in protein synthesis and in respiration but no change in levels of upstream and downstream mature transcripts

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2004.10.040

Confocal microscopy of the mitochondrial permeability transition in necrotic cell killing, apoptosis and autophagy

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2004.10.040

The anti-ageing effects of caloric restriction may involve stimulation of macroautophagy and lysosomal degradation, and can be intensified pharmacologically

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2004.10.040

10.1016/J.FEBSLET.2004.10.040

21 January 2018

Identifiers

DOI

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21 September 2017

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