(Q40546184)

21 September 2017

FGF2 inhibits proliferation and alters the cartilage-like phenotype of RCS cells (English)

1 reference

21 September 2017

4

1 reference

21 September 2017

Pavel Krejci

1

1 reference

21 September 2017

Vitezslav Bryja

2

1 reference

21 September 2017

Jiri Pachernik

3

1 reference

21 September 2017

Robert Pogue

5

1 reference

21 September 2017

Pertchoui Mekikian

6

1 reference

21 September 2017

William R Wilcox

7

1 reference

21 September 2017

language of work or name

English

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publication date

1 July 2004

1 reference

Experimental Cell Research

21 September 2017

1 reference

21 September 2017

297

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21 September 2017

152-164

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21 September 2017

1

1 reference

Mutations in the transmembrane domain of FGFR3 cause the most common genetic form of dwarfism, achondroplasia

21 September 2017

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

Thanatophoric dysplasia (types I and II) caused by distinct mutations in fibroblast growth factor receptor 3.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

Molecular, radiologic, and histopathologic correlations in thanatophoric dysplasia

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

Overexpression of FGFR3, Stat1, Stat5 and p21Cip1 correlates with phenotypic severity and defective chondrocyte differentiation in FGFR3-related chondrodysplasias

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

Activation of Stat1 by mutant fibroblast growth-factor receptor in thanatophoric dysplasia type II dwarfism

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

A Lys644Glu substitution in fibroblast growth factor receptor 3 (FGFR3) causes dwarfism in mice by activation of STATs and ink4 cell cycle inhibitors

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

Fibroblast growth factor inhibits chondrocytic growth through induction of p21 and subsequent inactivation of cyclin E-Cdk2

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

Differentiation plasticity of chondrocytes derived from mouse embryonic stem cells

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

Differential effects of fibroblast growth factor (FGF) 9 and FGF2 on proliferation, differentiation and terminal differentiation of chondrocytic cells in vitro

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

Use of a new rat chondrosarcoma cell line to delineate a 119-base pair chondrocyte-specific enhancer element and to define active promoter segments in the mouse pro-alpha 1(II) collagen gene

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

FGF signaling inhibits chondrocyte proliferation and regulates bone development through the STAT-1 pathway

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

Skeletal dysplasia and defective chondrocyte differentiation by targeted overexpression of fibroblast growth factor 9 in transgenic mice.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

Abnormal bone growth and selective translational regulation in basic fibroblast growth factor (FGF-2) transgenic mice

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

Specificity and mechanism of action of some commonly used protein kinase inhibitors

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

Activation of MAP kinase kinase is necessary and sufficient for PC12 differentiation and for transformation of NIH 3T3 cells

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

Cell cycle arrest mediated by the MEK/mitogen-activated protein kinase pathway

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

Ras promotes p21(Waf1/Cip1) protein stability via a cyclin D1-imposed block in proteasome-mediated degradation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

Acceleration of the G1/S phase transition by expression of cyclins D1 and E with an inducible system

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

Development of mice expressing a single D-type cyclin.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

FGF signaling targets the pRb-related p107 and p130 proteins to induce chondrocyte growth arrest

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

A network of transcriptional and signaling events is activated by FGF to induce chondrocyte growth arrest and differentiation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

Inhibitory effects of basic fibroblast growth factor on chondrocyte differentiation.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

Regulation of osteoblast, chondrocyte, and osteoclast functions by fibroblast growth factor (FGF)-18 in comparison with FGF-2 and FGF-10

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

Hypertrophic chondrocytes undergo further differentiation in culture

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

Fibroblast transgelin and smooth muscle SM22alpha are the same protein, the expression of which is down-regulated in many cell lines

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

The role of mesenchyme-like tissue in the pathogenesis of thanatophoric dysplasia

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

Signal transduction by fibroblast growth factor receptors

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

Mitogen-activated protein kinases p42mapk and p44mapk are required for fibroblast proliferation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

Fibroblast growth factor-2 has opposite effects on human breast cancer MCF-7 cell growth depending on the activation level of the mitogen-activated protein kinase pathway

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

Transient and sustained ERK phosphorylation and nuclear translocation in growth control

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

High-intensity Raf signal causes cell cycle arrest mediated by p21Cip1.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

Activation of the ERK1/2 and p38 mitogen-activated protein kinase pathways mediates fibroblast growth factor-induced growth arrest of chondrocytes

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

The retinoblastoma protein and cell cycle control

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

Overexpression of Cdk6-cyclin D3 highly sensitizes cells to physical and chemical transformation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

Cdk6-cyclin D3 complex evades inhibition by inhibitor proteins and uniquely controls cell's proliferation competence

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

Genetic alterations of cyclins, cyclin-dependent kinases, and Cdk inhibitors in human cancer

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

NGF regulates the PC12 cell cycle machinery through specific inhibition of the Cdk kinases and induction of cyclin D1

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

Cell surface, heparin-like molecules are required for binding of basic fibroblast growth factor to its high affinity receptor

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YEXCR.2004.03.011

10.1016/J.YEXCR.2004.03.011

7 January 2021

Identifiers

DOI

1 reference

21 September 2017

1 reference