(Q40576976)

English

Expression of dystrophin driven by the 1.35-kb MCK promoter ameliorates muscular dystrophy in fast, but not in slow muscles of transgenic mdx mice

scientific article published on July 2003

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scholarly article

1 reference

Europe PubMed Central

21 September 2017

Expression of dystrophin driven by the 1.35-kb MCK promoter ameliorates muscular dystrophy in fast, but not in slow muscles of transgenic mdx mice (English)

1 reference

Europe PubMed Central

21 September 2017

muscular dystrophy

1 reference

based on heuristic

inferred from title

7

1 reference

Europe PubMed Central

21 September 2017

Hanns Lochmüller

8

0 references

author name string

Patrick Dunant

1

1 reference

Europe PubMed Central

21 September 2017

Nancy Larochelle

2

1 reference

Europe PubMed Central

21 September 2017

Christian Thirion

3

1 reference

Europe PubMed Central

21 September 2017

Rolf Stucka

4

1 reference

Europe PubMed Central

21 September 2017

Daniel Ursu

5

1 reference

Europe PubMed Central

21 September 2017

Basil J Petrof

6

1 reference

Europe PubMed Central

21 September 2017

language of work or name

1 reference

based on heuristic

inferred from title

publication date

1 July 2003

1 reference

Europe PubMed Central

21 September 2017

Molecular Therapy

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Europe PubMed Central

21 September 2017

8

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Europe PubMed Central

21 September 2017

1

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Europe PubMed Central

21 September 2017

80-89

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Europe PubMed Central

21 September 2017

Molecular pathophysiology and targeted therapeutics for muscular dystrophy

1 reference

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Gene therapy: trials and tribulations

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Interferon-gamma inhibits transgene expression driven by SV40 or CMV promoters but augments expression driven by the mammalian MHC I promoter

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Immune responses to dystropin: implications for gene therapy of Duchenne muscular dystrophy

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Restricting expression prolongs expression of foreign genes introduced into animals by retroviruses

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Immune evasion by muscle-specific gene expression in dystrophic muscle

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Adeno-associated virus vector-mediated gene transfer into dystrophin-deficient skeletal muscles evokes enhanced immune response against the transgene product

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Designing gene therapy vectors: avoiding immune responses by using tissue-specific promoters

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In vivo expression of full-length human dystrophin from adenoviral vectors deleted of all viral genes

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Functional protection of dystrophic mouse (mdx) muscles after adenovirus-mediated transfer of a dystrophin minigene

1 reference

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Adenovirus-mediated dystrophin minigene transfer improves muscle strength in adult dystrophic (MDX) mice

1 reference

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Analysis of muscle creatine kinase regulatory elements in recombinant adenoviral vectors

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Adeno-associated virus vector carrying human minidystrophin genes effectively ameliorates muscular dystrophy in mdx mouse model

1 reference

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Viral vectors for gene transfer of micro-, mini-, or full-length dystrophin

1 reference

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Small-caliber skeletal muscle fibers do not suffer deleterious consequences of dystrophic gene expression

1 reference

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7 January 2021

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inferred from DOI database lookup

The short MCK1350 promoter/enhancer allows for sufficient dystrophin expression in skeletal muscles of mdx mice

1 reference

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7 January 2021

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Efficient muscle-specific transgene expression after adenovirus-mediated gene transfer in mice using a 1.35 kb muscle creatine kinase promoter/enhancer

1 reference

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Generation, validation, and large scale production of adenoviral recombinants with large size inserts such as a 6.3 kb human dystrophin cDNA.

1 reference

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Dystrophin protects the sarcolemma from stresses developed during muscle contraction

1 reference

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7 January 2021

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Three mouse models of muscular dystrophy: the natural history of strength and fatigue in dystrophin-, dystrophin/utrophin-, and laminin α2-deficient mice

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Viral vectors for gene therapy: the art of turning infectious agents into vehicles of therapeutics

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Expression of full-length and truncated dystrophin mini-genes in transgenic mdx mice

1 reference

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7 January 2021

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Very mild muscular dystrophy associated with the deletion of 46% of dystrophin

1 reference

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Modular flexibility of dystrophin: implications for gene therapy of Duchenne muscular dystrophy

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PCR and immunocytochemical analyses of dystrophin-positive fibers in Duchenne muscular dystrophy

1 reference

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Revertant fibres: a possible genetic therapy for Duchenne muscular dystrophy?

1 reference

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Massive idiosyncratic exon skipping corrects the nonsense mutation in dystrophic mouse muscle and produces functional revertant fibers by clonal expansion

1 reference

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Suppression of revertant fibers in mdx mice by expression of a functional dystrophin.

1 reference

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A chronic inflammatory response dominates the skeletal muscle molecular signature in dystrophin-deficient mdx mice

1 reference

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Helper (CD4(+)) and cytotoxic (CD8(+)) T cells promote the pathology of dystrophin-deficient muscle

1 reference

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The mdx mouse skeletal muscle myopathy: II. Contractile properties

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Tibialis anterior muscles in mdx mice are highly susceptible to contraction-induced injury

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Force and power output of fast and slow skeletal muscles from mdx mice 6-28 months old

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The creatine-creatine phosphate energy shuttle

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Intracellular compartmentation, structure and function of creatine kinase isoenzymes in tissues with high and fluctuating energy demands: the 'phosphocreatine circuit' for cellular energy homeostasis

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Effect of denervation on the distribution and developmental transition of phosphoglycerate mutase and creatine phosphokinase isozymes in rat muscles of different fiber-type composition.

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Profiles of creatine kinase isoenzyme compositions in single muscle fibres of different types.

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M-creatine kinase gene expression in mechanically overloaded skeletal muscle of transgenic mice

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Muscle creatine kinase sequence elements regulating skeletal and cardiac muscle expression in transgenic mice

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E-box sites and a proximal regulatory region of the muscle creatine kinase gene differentially regulate expression in diverse skeletal muscles and cardiac muscle of transgenic mice

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Mdx transgenic mouse: restoration of recombinant dystrophin to the dystrophic muscle

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1 reference

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Prevention of pathology in mdx mice by expression of utrophin: analysis using an inducible transgenic expression system.

1 reference

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1 reference

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Analysis of muscle creatine kinase gene regulatory elements in skeletal and cardiac muscles of transgenic mice

1 reference

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1 reference

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Mdx mice inducibly expressing dystrophin provide insights into the potential of gene therapy for duchenne muscular dystrophy

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DNA immunization utilizing a herpes simplex virus type 2 myogenic DNA vaccine protects mice from mortality and prevents genital herpes

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Differential expression and secretion of alpha 1 anti-trypsin between direct DNA injection and implantation of transfected myoblast

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Identifiers

10.1016/S1525-0016(03)00129-1

1 reference

Europe PubMed Central

21 September 2017

1 reference

Europe PubMed Central

21 September 2017

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