(Q40596368)

English

Protective effects of estradiol on TRAIL-induced apoptosis in a human oligodendrocytic cell line: evidence for multiple sites of interactions.

scientific article published on May 2004

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scholarly article

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17 November 2019

Protective effects of estradiol on TRAIL-induced apoptosis in a human oligodendrocytic cell line: evidence for multiple sites of interactions (English)

1 reference

Europe PubMed Central

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17 November 2019

6

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17 November 2019

Renato Bernardini

7

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17 November 2019

author name string

Cantarella G

1

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17 November 2019

Risuglia N

2

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17 November 2019

Lombardo G

3

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17 November 2019

Lempereur L

4

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17 November 2019

Nicoletti F

5

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17 November 2019

publication date

1 May 2004

1 reference

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17 November 2019

Cell Death & Differentiation

1 reference

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17 November 2019

11

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17 November 2019

5

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17 November 2019

503-511

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17 November 2019

https://scigraph.springernature.com/pub.10.1038/sj.cdd.4401367

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Apoptotic death of neurons exhibiting peripherin aggregates is mediated by the proinflammatory cytokine tumor necrosis factor-alpha

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Human brain-cell death induced by tumour-necrosis-factor-related apoptosis-inducing ligand (TRAIL)

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The receptor for the cytotoxic ligand TRAIL

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TRAIL-R2: a novel apoptosis-mediating receptor for TRAIL.

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An antagonist decoy receptor and a death domain-containing receptor for TRAIL

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7 January 2021

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Control of TRAIL-induced apoptosis by a family of signaling and decoy receptors

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The novel receptor TRAIL-R4 induces NF-kappaB and protects against TRAIL-mediated apoptosis, yet retains an incomplete death domain

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Lack of tumor necrosis factor-related apoptosis-inducing ligand but presence of its receptors in the human brain

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Neutralization of TRAIL death pathway protects human neuronal cell line from beta-amyloid toxicity

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TRAIL receptors 1 (DR4) and 2 (DR5) signal FADD-dependent apoptosis and activate NF-kappaB

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7 January 2021

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The death domain kinase RIP is essential for TRAIL (Apo2L)-induced activation of IkappaB kinase and c-Jun N-terminal kinase

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Estrogen receptor is expressed in different types of glial cells in culture.

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The involvement of reactive oxygen species (ROS) and p38 mitogen-activated protein (MAP) kinase in TRAIL/Apo2L-induced apoptosis

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7 January 2021

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TRAIL induces death of human oligodendrocytes isolated from adult brain

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A human glial hybrid cell line differentially expressing genes subserving oligodendrocyte and astrocyte phenotype

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Neuroprotective effects of estrogens: potential mechanisms of action

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Neurotrophic and neuroprotective actions of estrogens and their therapeutic implications

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FADD/MORT1 and caspase-8 are recruited to TRAIL receptors 1 and 2 and are essential for apoptosis mediated by TRAIL receptor 2

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Neuroprotective effects of estrogen against beta-amyloid toxicity are mediated by estrogen receptors in cultured neuronal cells

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Identifiers

10.1038/SJ.CDD.4401367

1 reference

Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:14739940%20AND%20SRC:MED&resulttype=core&format=json

17 November 2019

Dimensions Publication ID

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PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:14739940%20AND%20SRC:MED&resulttype=core&format=json

17 November 2019

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