(Q40646282)

22 September 2017

title

Epithelial Na+ channel mutants causing Liddle's syndrome retain ability to respond to aldosterone and vasopressin (English)

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22 September 2017

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4

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22 September 2017

Muriel Auberson

1

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22 September 2017

Nicole Hoffmann-Pochon

series ordinal

2

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22 September 2017

A Vandewalle

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3

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22 September 2017

Laurent Schild

series ordinal

5

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22 September 2017

publication date

20 May 2003

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American Journal of Physiology - Renal Physiology

22 September 2017

published in

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22 September 2017

volume

285

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22 September 2017

issue

3

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22 September 2017

page(s)

F459-71

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The serum and glucocorticoid kinase sgk increases the abundance of epithelial sodium channels in the plasma membrane of Xenopus oocytes

22 September 2017

cites work

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00071.2003

Effects of aldosterone on biosynthesis, traffic, and functional expression of epithelial sodium channels in A6 cells

21 January 2018

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https://api.crossref.org/works/10.1152%2FAJPRENAL.00071.2003

Amiloride-sensitive epithelial Na+ channel is made of three homologous subunits

21 January 2018

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Epithelial sodium channel regulated by aldosterone-induced protein sgk.

21 January 2018

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https://api.crossref.org/works/10.1152%2FAJPRENAL.00071.2003

Phosphorylation of Nedd4-2 by Sgk1 regulates epithelial Na(+) channel cell surface expression

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00071.2003

Cell-specific expression of epithelial sodium channel alpha, beta, and gamma subunits in aldosterone-responsive epithelia from the rat: localization by in situ hybridization and immunocytochemistry

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00071.2003

Vasopressin-mediated regulation of epithelial sodium channel abundance in rat kidney.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00071.2003

Cell surface expression of the epithelial Na channel and a mutant causing Liddle syndrome: a quantitative approach

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00071.2003

Activating mineralocorticoid receptor mutation in hypertension exacerbated by pregnancy

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00071.2003

A mutation causing pseudohypoaldosteronism type 1 identifies a conserved glycine that is involved in the gating of the epithelial sodium channel

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00071.2003

Hypertension caused by a truncated epithelial sodium channel gamma subunit: genetic heterogeneity of Liddle syndrome

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00071.2003

A de novo missense mutation of the beta subunit of the epithelial sodium channel causes hypertension and Liddle syndrome, identifying a proline-rich segment critical for regulation of channel activity

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00071.2003

Trafficking and cell surface stability of the epithelial Na+ channel expressed in epithelial Madin-Darby canine kidney cells

21 January 2018

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A novel mouse Nedd4 protein suppresses the activity of the epithelial Na+ channel

21 January 2018

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Molecular genetics of human blood pressure variation

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00071.2003

Aldosterone induces rapid apical translocation of ENaC in early portion of renal collecting system: possible role of SGK.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00071.2003

Effects of vasopressin and cAMP on single amiloride-blockable Na channels

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00071.2003

cAMP increases density of ENaC subunits in the apical membrane of MDCK cells in direct proportion to amiloride-sensitive Na(+) transport

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00071.2003

sgk is an aldosterone-induced kinase in the renal collecting duct. Effects on epithelial na+ channels

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00071.2003

Regulation of Na channels of the rat cortical collecting tubule by aldosterone.

21 January 2018

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https://api.crossref.org/works/10.1152%2FAJPRENAL.00071.2003

Production of high-titer helper-free retroviruses by transient transfection

21 January 2018

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https://api.crossref.org/works/10.1152%2FAJPRENAL.00071.2003

Transcriptome of a mouse kidney cortical collecting duct cell line: effects of aldosterone and vasopressin

21 January 2018

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Trafficking and cell surface stability of ENaC.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00071.2003

A mutation in the epithelial sodium channel causing Liddle disease increases channel activity in the Xenopus laevis oocyte expression system

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00071.2003

Identification of amino acid residues in the alpha, beta, and gamma subunits of the epithelial sodium channel (ENaC) involved in amiloride block and ion permeation

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00071.2003

Interactions of beta and gamma ENaC with Nedd4 can be facilitated by an ERK-mediated phosphorylation

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00071.2003

In vivo phosphorylation of the epithelial sodium channel

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00071.2003

The activity of the epithelial sodium channel is regulated by clathrin-mediated endocytosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00071.2003

Liddle's syndrome: heritable human hypertension caused by mutations in the beta subunit of the epithelial sodium channel

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00071.2003

Liddle's syndrome mutations disrupt cAMP-mediated translocation of the epithelial Na(+) channel to the cell surface

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00071.2003

Serum and glucocorticoid-regulated kinase modulates Nedd4-2-mediated inhibition of the epithelial Na+ channel

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00071.2003

Mechanism by which Liddle's syndrome mutations increase activity of a human epithelial Na+ channel

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00071.2003

Regulation of stability and function of the epithelial Na+ channel (ENaC) by ubiquitination

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00071.2003

Characterization of a novel protein-binding module--the WW domain

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00071.2003

Liddle disease caused by a missense mutation of beta subunit of the epithelial sodium channel gene

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00071.2003

Immortalized kidney epithelial cells as tools for hormonally regulated ion transport studies

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00071.2003

SGK integrates insulin and mineralocorticoid regulation of epithelial sodium transport

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00071.2003

10.1152/AJPRENAL.00071.2003

21 January 2018

Identifiers

DOI

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22 September 2017

1 reference