(Q40689433)

English

Hyperthermia-induced heat shock activates the transcription factor c/EBP-beta and augments IL-6 production in human intestinal epithelial cells

scientific article published on November 2002

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

22 September 2017

Hyperthermia-induced heat shock activates the transcription factor c/EBP-beta and augments IL-6 production in human intestinal epithelial cells (English)

1 reference

Europe PubMed Central

PubMed publication ID

22 September 2017

author name string

Eric S Hungness

1

1 reference

Europe PubMed Central

PubMed publication ID

22 September 2017

Bruce W Robb

2

1 reference

Europe PubMed Central

PubMed publication ID

22 September 2017

Guang-Ju Luo

3

1 reference

Europe PubMed Central

PubMed publication ID

22 September 2017

Dan D Hershko

4

1 reference

Europe PubMed Central

PubMed publication ID

22 September 2017

Per-Olof Hasselgren

5

1 reference

Europe PubMed Central

PubMed publication ID

22 September 2017

publication date

1 November 2002

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Europe PubMed Central

PubMed publication ID

22 September 2017

Journal of the American College of Surgeons

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Europe PubMed Central

PubMed publication ID

22 September 2017

195

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Europe PubMed Central

PubMed publication ID

22 September 2017

5

1 reference

Europe PubMed Central

PubMed publication ID

22 September 2017

619-626

1 reference

Europe PubMed Central

PubMed publication ID

22 September 2017

Role of the gut in multiple organ failure: bacterial translocation and permeability changes

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Mucosal production of complement C3 and serum amyloid A is differentially regulated in different parts of the gastrointestinal tract during endotoxemia in mice

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The pathophysiologic roles of interleukin-6 in human disease

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Interleukins and IgA synthesis. Human and murine interleukin 6 induce high rate IgA secretion in IgA-committed B cells

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IL-6 is an antiinflammatory cytokine required for controlling local or systemic acute inflammatory responses

1 reference

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Sepsis and endotoxemia stimulate intestinal interleukin-6 production*

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Endotoxemia and IL-1 beta stimulate mucosal IL-6 production in different parts of the gastrointestinal tract

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Endotoxin stimulates interleukin-6 production in intestinal epithelial cells. A synergistic effect with prostaglandin E2.

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Interleukin-6 production in human intestinal epithelial cells increases in association with the heat shock response

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Signal transduction by tumor necrosis factor and gene regulation of the inflammatory cytokine interleukin-6.

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Induction of the stress response in vivo decreases nuclear factor-kappa B activity in jejunal mucosa of endotoxemic mice

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IL-1beta activates C/EBP-beta and delta in human enterocytes through a mitogen-activated protein kinase signaling pathway

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CCAAT/enhancer-binding proteins are mediators in the protein kinase A-dependent activation of the decidual prolactin promoter

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Single-Step Method of RNA Isolation by Acid Guanidinium Thiocyanate–Phenol–Chloroform Extraction

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A liver-enriched transcriptional activator protein, LAP, and a transcriptional inhibitory protein, LIP, are translated from the same mRNA

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CCAAT/enhancer-binding protein mRNA is translated into multiple proteins with different transcription activation potentials

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Transcription factors C/EBP-beta and -delta regulate IL-6 production in IL-1beta-stimulated human enterocytes

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Evidence that a rapidly turning over protein, normally degraded by proteasomes, regulates hsp72 gene transcription in HepG2 cells

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Proteasome inhibition leads to a heat-shock response, induction of endoplasmic reticulum chaperones, and thermotolerance

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Proteasome inhibition leads to the activation of all members of the heat-shock-factor family

1 reference

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7 January 2021

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Proteasome inhibitors induce heat shock response and increase IL-6 expression in human intestinal epithelial cells

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Induction of a subgroup of acute phase protein genes in mouse liver by hyperthermia

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Serum Cytokine Levels in Human Septic Shock

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Identifiers

10.1016/S1072-7515(02)01342-X

1 reference

Europe PubMed Central

PubMed publication ID

22 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

22 September 2017

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