(Q40733537)

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Cytokine-induced apoptosis and necrosis are preceded by disruption of the mitochondrial membrane potential (Deltapsi(m)) in pancreatic RINm5F cells: prevention by Bcl-2.

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Europe PubMed Central

PubMed publication ID

23 September 2017

Cytokine-induced apoptosis and necrosis are preceded by disruption of the mitochondrial membrane potential (Deltapsi(m)) in pancreatic RINm5F cells: prevention by Bcl-2. (English)

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Europe PubMed Central

PubMed publication ID

23 September 2017

apoptotic process

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author name string

Andreea Barbu

1

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

Nils Welsh

2

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Europe PubMed Central

PubMed publication ID

23 September 2017

Johan Saldeen

3

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Europe PubMed Central

PubMed publication ID

23 September 2017

publication date

1 April 2002

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Europe PubMed Central

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23 September 2017

Molecular and Cellular Endocrinology

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Europe PubMed Central

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23 September 2017

190

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Europe PubMed Central

PubMed publication ID

23 September 2017

1-2

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Europe PubMed Central

PubMed publication ID

23 September 2017

75-82

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Europe PubMed Central

PubMed publication ID

23 September 2017

Modulation of the mitochondrial permeability transition by nitric oxide

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The permeability transition pore. Control points of a cyclosporin A-sensitive mitochondrial channel involved in cell death

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Suppression of apoptosis in a cytotoxic T-cell line by interleukin 2-mediated gene transcription and deregulated expression of the protooncogene bcl-2.

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Suppression of apoptosis by nitric oxide via inhibition of interleukin-1beta-converting enzyme (ICE)-like and cysteine protease protein (CPP)-32-like proteases

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Bcl-2 protects against FCCP-induced apoptosis and mitochondrial membrane potential depolarization in PC12 cells

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Shift of the cellular oxidation-reduction potential in neural cells expressing Bcl-2.

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Bcl-2 and mitochondrial oxygen radicals. New approaches with reactive oxygen species-sensitive probes.

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Reactive oxygen species (ROS) mediates the mitochondrial-dependent apoptosis induced by transforming growth factor (beta) in fetal hepatocytes.

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Nitric oxide induces apoptosis via triggering mitochondrial permeability transition.

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Establishment of mouse cell lines which constitutively secrete large quantities of interleukin 2, 3, 4 or 5, using modified cDNA expression vectors

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Elevation of resting mitochondrial membrane potential of neural cells by cyclosporin A, BAPTA-AM, and bcl-2.

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Bcl-2 prevents mitochondrial permeability transition and cytochrome c release via maintenance of reduced pyridine nucleotides.

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Mitochondrial control of apoptosis

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The mitochondrial death/life regulator in apoptosis and necrosis

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Intracellular adenosine triphosphate (ATP) concentration: a switch in the decision between apoptosis and necrosis

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Evaluation of fluorescent dyes for the detection of mitochondrial membrane potential changes in cultured cardiomyocytes

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Apoptosis and the pathogenesis of IDDM: a question of life and death

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Bcl-2 prevents Bax oligomerization in the mitochondrial outer membrane

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Peroxynitrite causes calcium efflux from mitochondria which is prevented by Cyclosporin A.

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Cytotoxic effects of cytokines on rat islets: evidence for involvement of free radicals and lipid peroxidation

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Interleukin-1 beta induced activation of NF-kappa B in insulin producing RINm5F cells is prevented by the protease inhibitor N alpha-p-tosyl-L-lysine chloromethylketone

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Nicotinamide-induced apoptosis in insulin producing cells is associated with cleavage of poly(ADP-ribose) polymerase

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The acid sphingomyelinase inhibitor SR33557 counteracts TNF-alpha-mediated potentiation of IL-1beta-induced NF-kappaB activation in the insulin-producing cell line Rinm5F.

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Caspases: intracellular signaling by proteolysis

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Metabolism and beta-cell function of rat pancreatic islets exposed to human interleukin-1 beta in the presence of a high glucose concentration.

1 reference

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Two CD95 (APO-1/Fas) signaling pathways

1 reference

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Bcl-2 prevents apoptotic mitochondrial dysfunction by regulating proton flux

1 reference

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Oxygen reduction by nitric-oxide synthases

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In vivo visualization of oxygen radical-dependent photoemission

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Interleukin-lβ-Induced Nitric Oxide Production in Isolated Rat Pancreatic Islets Requires Gene Transcription and May Lead to Inhibition of the Krebs Cycle Enzyme Aconitase*

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Changes in the generation of reactive oxygen species and in mitochondrial membrane potential during apoptosis induced by the antidepressants imipramine, clomipramine, and citalopram and the effects on these changes by Bcl-2 and Bcl-X(L)

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Identifiers

10.1016/S0303-7207(02)00009-6

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

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