(Q40770217)

English

Gliotoxin induces apoptosis in cultured macrophages via production of reactive oxygen species and cytochrome c release without mitochondrial depolarization

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

Gliotoxin induces apoptosis in cultured macrophages via production of reactive oxygen species and cytochrome c release without mitochondrial depolarization (English)

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

0 references

apoptotic process

0 references

reactive oxygen species

1 reference

based on heuristic

inferred from title

author name string

Suen YK

1

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

Fung KP

2

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

Lee CY

3

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

Kong SK

4

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

publication date

1 July 2001

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

Free Radical Research

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

35

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

1

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

1-10

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

Gliotoxin and related epipolythiodioxopiperazines

1 reference

https://api.crossref.org/works/10.1080%2F10715760100300541

21 January 2018

Hydrogen peroxide-induced apoptosis in HL-60 cells requires caspase-3 activation.

1 reference

https://api.crossref.org/works/10.1080%2F10715760100300541

21 January 2018

The mitochondrial death/life regulator in apoptosis and necrosis

1 reference

https://api.crossref.org/works/10.1080%2F10715760100300541

21 January 2018

Biochemical pathways of caspase activation during apoptosis

1 reference

https://api.crossref.org/works/10.1080%2F10715760100300541

21 January 2018

Slow rise of Ca2+ and slow release of reactive oxygen species are two cross-talked events important in tumour necrosis factor-alpha-mediated apoptosis

1 reference

https://api.crossref.org/works/10.1080%2F10715760100300541

21 January 2018

Concanavalin A induced apoptosis in murine macrophage PU5-1.8 cells through clustering of mitochondria and release of cytochrome c.

1 reference

https://api.crossref.org/works/10.1080%2F10715760100300541

21 January 2018

Sequential reduction of mitochondrial transmembrane potential and generation of reactive oxygen species in early programmed cell death

1 reference

https://api.crossref.org/works/10.1080%2F10715760100300541

21 January 2018

Cytotoxicity and apoptosis produced by arachidonic acid in Hep G2 cells overexpressing human cytochrome P4502E1.

1 reference

https://api.crossref.org/works/10.1080%2F10715760100300541

21 January 2018

Changes in intramitochondrial and cytosolic pH: early events that modulate caspase activation during apoptosis

1 reference

https://api.crossref.org/works/10.1080%2F10715760100300541

21 January 2018

JC-1, but not DiOC6(3) or rhodamine 123, is a reliable fluorescent probe to assess delta psi changes in intact cells: implications for studies on mitochondrial functionality during apoptosis

1 reference

https://api.crossref.org/works/10.1080%2F10715760100300541

21 January 2018

Bax and adenine nucleotide translocator cooperate in the mitochondrial control of apoptosis

1 reference

https://api.crossref.org/works/10.1080%2F10715760100300541

21 January 2018

Outer mitochondrial membrane permeability can regulate coupled respiration and cell survival

1 reference

https://api.crossref.org/works/10.1080%2F10715760100300541

21 January 2018

Gliotoxin-induced cytotoxicity proceeds via apoptosis and is mediated by caspases and reactive oxygen species in LLC-PK1 cells

1 reference

https://api.crossref.org/works/10.1080%2F10715760100300541

21 January 2018

Mitochondrial cytochrome c release in apoptosis occurs upstream of DEVD-specific caspase activation and independently of mitochondrial transmembrane depolarization.

1 reference

https://api.crossref.org/works/10.1080%2F10715760100300541

21 January 2018

Collapse of the inner mitochondrial transmembrane potential is not required for apoptosis of HL60 cells.

1 reference

https://api.crossref.org/works/10.1080%2F10715760100300541

21 January 2018

Identifiers

10.1080/10715760100300541

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

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