(Q40772134)

23 September 2017

Effects of overexpression of the SH2-containing inositol phosphatase SHIP on proliferation and apoptosis of erythroid AS-E2 cells (English)

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23 September 2017

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A K Boer

1

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23 September 2017

A L Drayer

2

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23 September 2017

E Vellenga

3

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23 September 2017

A-K Boer

1

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AL Drayer

2

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publication date

1 November 2001

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23 September 2017

November 2001

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published in

Leukemia

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23 September 2017

15

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23 September 2017

11

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23 September 2017

1750-1757

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https://scigraph.springernature.com/pub.10.1038/sj.leu.2402261

23 September 2017

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cites work

Generation of committed erythroid BFU-E and CFU-E progenitors does not require erythropoietin or the erythropoietin receptor

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402261

7 January 2021

STAT5 as a molecular regulator of proliferation, differentiation and apoptosis in hematopoietic cells

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402261

7 January 2021

Cloning and expression of a human placenta inositol 1,3,4,5-tetrakisphosphate and phosphatidylinositol 3,4,5-trisphosphate 5-phosphatase

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402261

7 January 2021

The 145-kDa protein induced to associate with Shc by multiple cytokines is an inositol tetraphosphate and phosphatidylinositol 3,4,5-triphosphate 5-phosphatase

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402261

7 January 2021

p150Ship, a signal transduction molecule with inositol polyphosphate-5-phosphatase activity

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402261

7 January 2021

Multiple forms of an inositol polyphosphate 5-phosphatase form signaling complexes with Shc and Grb2

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402261

7 January 2021

The Src homology 2 (SH2) domain of SH2-containing inositol phosphatase (SHIP) is essential for tyrosine phosphorylation of SHIP, its association with Shc, and its induction of apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402261

7 January 2021

The phosphatidylinositol polyphosphate 5-phosphatase SHIP and the protein tyrosine phosphatase SHP-2 form a complex in hematopoietic cells which can be regulated by BCR/ABL and growth factors

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402261

7 January 2021

Interleukin-3 induces the association of the inositol 5-phosphatase SHIP with SHP2

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402261

7 January 2021

The SH2 domain-containing inositol 5'-phosphatase (SHIP) recruits the p85 subunit of phosphoinositide 3-kinase during FcgammaRIIb1-mediated inhibition of B cell receptor signaling

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402261

7 January 2021

The inositol polyphosphate 5-phosphatase ship is a crucial negative regulator of B cell antigen receptor signaling

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402261

7 January 2021

Role of the inositol phosphatase SHIP in negative regulation of the immune system by the receptor Fc(gamma)RIIB.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402261

7 January 2021

Transient translocation of the B cell receptor and Src homology 2 domain-containing inositol phosphatase to lipid rafts: evidence toward a role in calcium regulation.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402261

7 January 2021

Tyrosine phosphorylation and relocation of SHIP are integrin-mediated in thrombin-stimulated human blood platelets

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402261

7 January 2021

Targeted disruption of SHIP leads to Steel factor-induced degranulation of mast cells.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402261

7 January 2021

An SH2 domain-containing 5' inositolphosphatase inhibits insulin-induced GLUT4 translocation and growth factor-induced actin filament rearrangement

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402261

7 January 2021

Altered responsiveness to chemokines due to targeted disruption of SHIP.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402261

7 January 2021

Targeted disruption of SHIP leads to hemopoietic perturbations, lung pathology, and a shortened life span

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402261

7 January 2021

SHIP is a negative regulator of growth factor receptor-mediated PKB/Akt activation and myeloid cell survival

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402261

7 January 2021

The inositol phosphatase SHIP inhibits Akt/PKB activation in B cells

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402261

7 January 2021

The src homology 2-containing inositol phosphatase (SHIP) is the gatekeeper of mast cell degranulation

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402261

7 January 2021

Establishment and characterization of a new erythropoietin-dependent acute myeloid leukemia cell line, AS-E2.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402261

7 January 2021

Post-translational modification of human brain type I inositol-1,4,5-trisphosphate 5-phosphatase by farnesylation

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402261

7 January 2021

Reactive Oxygen Species Regulate Activation-Induced T Cell Apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402261

7 January 2021

Mutation of the conserved domains of two inositol polyphosphate 5-phosphatases

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402261

7 January 2021

The Ras/phosphatidylinositol 3-kinase and Ras/ERK pathways function as independent survival modules each of which inhibits a distinct apoptotic signaling pathway in sympathetic neurons.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402261

7 January 2021

Fas antigen (CD95) in pure erythroid cell line AS-E2 is induced by interferon-gamma and tumor necrosis factor-alpha and potentiates apoptotic death

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402261

7 January 2021

SHIP-mediated inhibition of K562 erythroid differentiation requires an intact catalytic domain and Shc binding site

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402261

7 January 2021

Specificity of receptor tyrosine kinase signaling: transient versus sustained extracellular signal-regulated kinase activation

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402261

7 January 2021

Mechanisms and control of programmed cell death in invertebrates.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402261

7 January 2021

Essential role for the C-terminal noncatalytic region of SHIP in FcgammaRIIB1-mediated inhibitory signaling

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402261

7 January 2021

Identifiers

DOI

10.1038/SJ.LEU.2402261

0 references

1 reference