(Q40817283)

English

Transient activation of Jun N-terminal kinases and protection from apoptosis by the insulin-like growth factor I receptor can be suppressed by dicumarol

scientific article published on 21 February 2001

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

Transient activation of Jun N-terminal kinases and protection from apoptosis by the insulin-like growth factor I receptor can be suppressed by dicumarol (English)

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

author name string

D Krause

1

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

A Lyons

2

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

C Fennelly

3

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

R O'Connor

4

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

language of work or name

0 references

publication date

21 February 2001

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

Journal of Biological Chemistry

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Europe PubMed Central

PubMed publication ID

24 September 2017

276

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

22

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

19244-19252

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

Regulation of neuronal survival by the serine-threonine protein kinase Akt

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

Effect of a null mutation of the insulin-like growth factor I receptor gene on growth and transformation of mouse embryo fibroblasts

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

Antisense-mediated reduction in insulin-like growth factor-I receptor expression suppresses the malignant phenotype of a human alveolar rhabdomyosarcoma

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

Suppression of insulin-like growth factor type I receptor by a triple-helix strategy inhibits IGF-I transcription and tumorigenic potential of rat C6 glioblastoma cells

1 reference

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21 January 2018

Suppression of c-Myc-induced apoptosis by Ras signalling through PI(3)K and PKB.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

Akt promotes cell survival by phosphorylating and inhibiting a Forkhead transcription factor

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

CREB is a regulatory target for the protein kinase Akt/PKB

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

Regulation of cell death protease caspase-9 by phosphorylation

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

Akt phosphorylation of BAD couples survival signals to the cell-intrinsic death machinery

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

Cell survival promoted by the Ras-MAPK signaling pathway by transcription-dependent and -independent mechanisms

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

Multiple signaling pathways of the insulin-like growth factor 1 receptor in protection from apoptosis

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

Negative regulation of PKB/Akt-dependent cell survival by the tumor suppressor PTEN

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

New insights into tumor suppression: PTEN suppresses tumor formation by restraining the phosphoinositide 3-kinase/AKT pathway

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

Akt-dependent and -independent survival signaling pathways utilized by insulin-like growth factor I.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

Identification of domains of the insulin-like growth factor I receptor that are required for protection from apoptosis

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

Tyrosine residues in the C-terminal domain of the insulin-like growth factor-I receptor mediate mitogenic and tumorigenic signals

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

The Bcr-Abl leukemia oncogene activates Jun kinase and requires Jun for transformation

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

Activation of the JNK pathway is essential for transformation by the Met oncogene

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

JNK is required for effector T-cell function but not for T-cell activation

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

Withdrawal of survival factors results in activation of the JNK pathway in neuronal cells leading to Fas ligand induction and cell death

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

Requirement of JNK for stress-induced activation of the cytochrome c-mediated death pathway

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

Early activation of c-Jun N-terminal kinase and p38 kinase regulate cell survival in response to tumor necrosis factor alpha

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

Quinone reductase inhibitors block SAPK/JNK and NFkappaB pathways and potentiate apoptosis

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

Bidirectional regulation of p38 kinase and c-Jun N-terminal protein kinase by insulin-like growth factor-I.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

Prolonged activation of ERK2 by epidermal growth factor and other growth factors requires a functional insulin-like growth factor 1 receptor

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

The stress-activated protein kinase pathway mediates cell death following injury induced by cis-platinum, UV irradiation or heat

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

New role for Shc in activation of the phosphatidylinositol 3-kinase/Akt pathway

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

Anisomycin-activated protein kinases p45 and p55 but not mitogen-activated protein kinases ERK-1 and -2 are implicated in the induction of c-fos and c-jun

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

Insulin-like growth factor physiology and cancer risk

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

The stress-activated protein kinase pathways

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

Stability of the ATF2 transcription factor is regulated by phosphorylation and dephosphorylation

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

Oncogenic and transcriptional cooperation with Ha-Ras requires phosphorylation of c-Jun on serines 63 and 73

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

The Jun kinase/stress-activated protein kinase pathway functions to regulate DNA repair and inhibition of the pathway sensitizes tumor cells to cisplatin

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

c-Jun regulates cell cycle progression and apoptosis by distinct mechanisms

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

The c-Jun NH(2)-terminal kinase promotes insulin resistance during association with insulin receptor substrate-1 and phosphorylation of Ser(307)

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

c-Jun NH(2)-terminal kinase inhibits targeting of the protein phosphatase calcineurin to NFATc1.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

Insulin-like growth factor-I induces bcl-2 promoter through the transcription factor cAMP-response element-binding protein.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

Interaction in vitro of the product of the c-Crk-II proto-oncogene with the insulin-like growth factor I receptor

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

PSK, a novel STE20-like kinase derived from prostatic carcinoma that activates the c-Jun N-terminal kinase mitogen-activated protein kinase pathway and regulates actin cytoskeletal organization

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

Src and Cas mediate JNK activation but not ERK1/2 and p38 kinases by reactive oxygen species

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

Interaction of hematopoietic progenitor kinase 1 with adapter proteins Crk and CrkL leads to synergistic activation of c-Jun N-terminal kinase

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

A novel human STE20-related protein kinase, HGK, that specifically activates the c-Jun N-terminal kinase signaling pathway

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008186200

21 January 2018

Identifiers

10.1074/JBC.M008186200

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

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