(Q40827710)

24 September 2017

Bcr-Abl has a greater intrinsic capacity than v-Abl to induce the neoplastic expansion of myeloid cells (English)

1 reference

24 September 2017

Gross AW

1

1 reference

24 September 2017

Ren R

series ordinal

2

1 reference

24 September 2017

language of work or name

English

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publication date

1 December 2000

1 reference

24 September 2017

1 reference

24 September 2017

19

1 reference

24 September 2017

6286-6296

1 reference

24 September 2017

54

1 reference

https://scigraph.springernature.com/pub.10.1038/sj.onc.1204023

24 September 2017

0 references

cites work

A clonogenic common myeloid progenitor that gives rise to all myeloid lineages

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204023

Commitment to the B lymphoid lineage occurs before DH-JH recombination

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204023

Tyrosyl phosphorylation and DNA binding activity of signal transducers and activators of transcription (STAT) proteins in hematopoietic cell lines transformed by Bcr/Abl

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204023

Induction of chronic myelogenous leukemia in mice by the P210bcr/abl gene of the Philadelphia chromosome

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204023

Direct interaction of Jak1 and v-Abl is required for v-Abl-induced activation of STATs and proliferation

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204023

Jak-STAT signaling induced by the v-abl oncogene

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204023

Hematologic disease induced in BALB/c mice by a bcr-abl retrovirus is influenced by the infection conditions

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204023

The biology of chronic myeloid leukemia

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204023

Bcr-Abl with an SH3 deletion retains the ability To induce a myeloproliferative disease in mice, yet c-Abl activated by an SH3 deletion induces only lymphoid malignancy

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204023

Tissue-regulated differentiation and maturation of a v-abl-immortalized mast cell-committed progenitor

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204023

Resolution and characterization of pro-B and pre-pro-B cell stages in normal mouse bone marrow

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204023

Acute leukaemia in bcr/abl transgenic mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204023

P210 and P190(BCR/ABL) induce the tyrosine phosphorylation and DNA binding activity of multiple specific STAT family members

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204023

Induction of a chronic myelogenous leukemia-like syndrome in mice with v-abl and BCR/ABL.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204023

Identification of clonogenic common lymphoid progenitors in mouse bone marrow

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204023

The P190, P210, and P230 forms of the BCR/ABL oncogene induce a similar chronic myeloid leukemia-like syndrome in mice but have different lymphoid leukemogenic activity

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204023

Production of high-titer helper-free retroviruses by transient transfection

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204023

A quantitative assay for transformation of bone marrow cells by Abelson murine leukemia virus

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204023

21 January 2018

Chronic myeloid leukemia

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204023

v-abl causes hematopoietic disease distinct from that caused by bcr-abl

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204023

Fetal anemia and apoptosis of red cell progenitors in Stat5a-/-5b-/- mice: a direct role for Stat5 in Bcl-X(L) induction

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204023

Integrin-mediated regulation of hematopoiesis: do BCR/ABL-induced defects in integrin function underlie the abnormal circulation and proliferation of CML progenitors?

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204023

Pathophysiology of CML: do defects in integrin function contribute to the premature circulation and massive expansion of the BCR/ABL positive clone?

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204023

Signaling pathways activated by oncogenic forms of Abl tyrosine kinase

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204023

bcr-abl, the hallmark of chronic myeloid leukaemia in man, induces multiple haemopoietic neoplasms in mice.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204023

inferred from DOI database lookup

7 January 2021

based on heuristic

Signal transduction by wild-type and leukemogenic Abl proteins.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204023

inferred from DOI database lookup

7 January 2021

based on heuristic

The viral and cellular forms of the Abelson (abl) oncogene

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204023

inferred from DOI database lookup

7 January 2021

based on heuristic

Identifiers

DOI

10.1038/SJ.ONC.1204023

1 reference

Dimensions Publication ID

24 September 2017

0 references

1 reference