(Q40844250)

English

Suppression of experimental autoimmune myasthenia gravis in IL-10 gene-disrupted mice is associated with reduced B cells and serum cytotoxicity on mouse cell line expressing AChR.

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scholarly article

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Europe PubMed Central

PubMed publication ID

24 September 2017

Suppression of experimental autoimmune myasthenia gravis in IL-10 gene-disrupted mice is associated with reduced B cells and serum cytotoxicity on mouse cell line expressing AChR. (English)

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

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myasthenia gravis

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author name string

Poussin MA

1

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

Goluszko E

2

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

Hughes TK

3

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

Duchicella SI

4

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

Christadoss P

5

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

publication date

1 November 2000

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Europe PubMed Central

PubMed publication ID

24 September 2017

Journal of Neuroimmunology

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Europe PubMed Central

PubMed publication ID

24 September 2017

111

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

1-2

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Europe PubMed Central

PubMed publication ID

24 September 2017

152-160

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

Serum globulin in myasthenia gravis: inhibition of alpha-bungarotoxin binding to acetylcholine receptors

1 reference

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Mechanism of enhanced susceptibility to sepsis following hemorrhage. Interleukin-10 suppression of T-cell response is mediated by eicosanoid-induced interleukin-4 release.

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IL-10 suppresses experimental autoimmune neuritis and down-regulates TH1-type immune responses

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Interferon gamma (IFN-gamma) is necessary for the genesis of acetylcholine receptor-induced clinical experimental autoimmune myasthenia gravis in mice

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Interleukin 10 inhibits allogeneic proliferative and cytotoxic T cell responses generated in primary mixed lymphocyte cultures

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Human interleukin 10 induces naive surface immunoglobulin D+ (sIgD+) B cells to secrete IgG1 and IgG3

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B-cell-derived IL-10: production and function

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Interleukin-10 differentially modulates MHC class II expression by mesangial cells and macrophages in vitro and in vivo

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Animal models of myasthenia gravis

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The effect of B cell deficiency on the immune response to acetylcholine receptor and the development of experimental autoimmune myasthenia gravis

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Effect of myasthenic patients' immunoglobulin on acetylcholine receptor turnover: selectivity of degradation process

1 reference

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Neuromuscular Junction in Myasthenia Gravis: Decreased Acetylcholine Receptors

1 reference

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Modulation of acetylcholine receptor by antibody against the receptor

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Precursor B cells for autoantibody production in genomically Fas-intact autoimmune disease are not subject to Fas-mediated immune elimination

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Markers in the promoter region of interleukin-10 (IL-10) gene in myasthenia gravis: implications of diverse effects of IL-10 in the pathogenesis of the disease

1 reference

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based on heuristic

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Myasthenic immunoglobulin accelerates acetylcholine receptor degradation

1 reference

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Interleukin-4 deficiency facilitates development of experimental myasthenia gravis and precludes its prevention by nasal administration of CD4+ epitope sequences of the acetylcholine receptor

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Involvement of both major histocompatibility complex class II alpha and beta chains in CD4 function indicates a role for ordered oligomerization in T cell activation

1 reference

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Interleukin-10-deficient mice develop chronic enterocolitis

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Measurement of cell numbers by means of the endogenous enzyme hexosaminidase. Applications to detection of lymphokines and cell surface antigens

1 reference

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7 January 2021

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Experimental autoimmune myasthenia gravis induction in B cell-deficient mice

1 reference

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7 January 2021

based on heuristic

inferred from DOI database lookup

29 Production and assay of antibodies to acetylcholine receptors

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Role of interleukin 10 in the B lymphocyte hyperactivity and autoantibody production of human systemic lupus erythematosus

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7 January 2021

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inferred from DOI database lookup

Mucosal tolerance to experimental autoimmune myasthenia gravis is associated with down-regulation of AChR-specific IFN-gamma-expressing Th1-like cells and up-regulation of TGF-beta mRNA in mononuclear cells

1 reference

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based on heuristic

inferred from DOI database lookup

Tumor necrosis factor-alpha, lymphotoxin, interleukin (IL)-6, IL-10, IL-12 and perforin mRNA expression in mononuclear cells in response to acetylcholine receptor is augmented in myasthenia gravis

1 reference

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7 January 2021

based on heuristic

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IL-12 is involved in the induction of experimental autoimmune myasthenia gravis, an antibody-mediated disease

1 reference

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Cellular mRNA expression of interferon-gamma (IFN-gamma), IL-4 and IL-10 relates to resistance to experimental autoimmune myasthenia gravis (EAMG) in young Lewis rats

1 reference

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IL-10 selectively regulates murine Ig isotype switching

1 reference

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based on heuristic

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Interleukin-12 enhances clinical experimental autoimmune myasthenia gravis in susceptible but not resistant mice

1 reference

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based on heuristic

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IL-12 directly stimulates expression of IL-10 by CD5+ B cells and IL-6 by both CD5+ and CD5- B cells: possible involvement in age-associated cytokine dysregulation

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Interleukin-12 and -10 and gamma interferon regulate polyclonal and ligand-specific expansion of murine B-1 cells.

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Acetylcholine receptor-reactive T cells in myasthenia gravis: evidence for the involvement of different subpopulations of T helper cells

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Both CD4+ and CD8+ T cells are essential to induce experimental autoimmune myasthenia gravis

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Linomide suppresses both Th1 and Th2 cytokines in experimental autoimmune myasthenia gravis

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Identifiers

10.1016/S0165-5728(00)00385-4

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

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