(Q40865275)

English

Cooperation between AP1 and PEA3 sites within the progression elevated gene-3 (PEG-3) promoter regulate basal and differential expression of PEG-3 during progression of the oncogenic phenotype in transformed rat embryo cells

scientific article published on July 2000

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scholarly article

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Europe PubMed Central

24 September 2017

Cooperation between AP1 and PEA3 sites within the progression elevated gene-3 (PEG-3) promoter regulate basal and differential expression of PEG-3 during progression of the oncogenic phenotype in transformed rat embryo cells (English)

1 reference

Europe PubMed Central

24 September 2017

author name string

Su Z

1

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Europe PubMed Central

24 September 2017

Shi Y

2

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Europe PubMed Central

24 September 2017

Fisher PB

3

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Europe PubMed Central

24 September 2017

language of work or name

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publication date

1 July 2000

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Europe PubMed Central

24 September 2017

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Europe PubMed Central

24 September 2017

19

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Europe PubMed Central

24 September 2017

3411-3421

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Europe PubMed Central

24 September 2017

30

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Europe PubMed Central

24 September 2017

https://scigraph.springernature.com/pub.10.1038/sj.onc.1203666

0 references

Reversibility of progression of the transformed phenotype in Ad5-transformed rat embryo cells

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1203666

21 January 2018

Molecular themes in oncogenesis

1 reference

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21 January 2018

Specificities of protein-protein and protein-DNA interaction of GABP alpha and two newly defined ets-related proteins

2 references

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21 January 2018

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Accurate transcription initiation by RNA polymerase II in a soluble extract from isolated mammalian nuclei

1 reference

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21 January 2018

Cooperation of two PEA3/AP1 sites in uPA gene induction by TPA and FGF-2.

1 reference

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Suppression of the progression phenotype in somatic cell hybrids occurs in the absence of altered adenovirus type 5 gene expression

1 reference

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Involvement of AP1 and PEA3 binding sites in the regulation of murine tissue inhibitor of metalloproteinases-1 (TIMP-1) transcription

1 reference

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21 January 2018

Interactions between adenovirus, a tumor promoter, and chemical carcinogens in transformation of rat embryo cell cultures

1 reference

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Tumour promoters enhance anchorage-independent growth of adenovirus-transformed cells without altering the integration pattern of viral sequences

1 reference

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Tumor promoters and epidermal growth factor stimulate anchorage-independent growth of adenovirus-transformed rat embryo cells

1 reference

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Analysis of type 5 adenovirus transformation with a cloned rat embryo cell line (CREF)

1 reference

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Use of the human EF-1alpha promoter for expression can significantly increase success in establishing stable cell lines with consistent expression: a study using the tetracycline-inducible system in human cancer cells

1 reference

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Cell cycle control and cancer

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Reciprocal subtraction differential RNA display: an efficient and rapid procedure for isolating differentially expressed gene sequences

1 reference

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AP-1 function and regulation

1 reference

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Antioncogenes and human cancer

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The tumor suppressor genes

1 reference

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Cancer metastasis and angiogenesis: An imbalance of positive and negative regulation

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Stromal-epithelial interaction mediates steroidal regulation of metalloproteinase expression in human endometrium

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The ets gene family

1 reference

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A dominant negative to activation protein-1 (AP1) that abolishes DNA binding and inhibits oncogenesis.

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Interleukin-1 or phorbol induction of the stromelysin promoter requires an element that cooperates with AP-1.

1 reference

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Regulation of urokinase-type plasminogen activator gene transcription by macrophage colony-stimulating factor.

1 reference

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21 January 2018

Subtraction hybridization identifies a transformation progression-associated gene PEG-3 with sequence homology to a growth arrest and DNA damage-inducible gene

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1203666

21 January 2018

PEG-3, a nontransforming cancer progression gene, is a positive regulator of cancer aggressiveness and angiogenesis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1203666

21 January 2018

The multistep nature of cancer

1 reference

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The Ets family of transcription factors

1 reference

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The role of Jun, Fos and the AP-1 complex in cell-proliferation and transformation

1 reference

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7 January 2021

based on heuristic

inferred from DOI database lookup

Identifiers

10.1038/SJ.ONC.1203666

1 reference

Europe PubMed Central

24 September 2017

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1 reference

Europe PubMed Central

24 September 2017

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