(Q40876083)

24 September 2017

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title

Polycystic kidney disease: primary extracellular matrix abnormality or defective cellular differentiation? (English)

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polycystic kidney disease

24 September 2017

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Calvet JP

1

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PubMed ID

8433548

24 September 2017

language of work or name

English

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publication date

1 January 1993

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24 September 2017

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24 September 2017

43

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24 September 2017

101-108

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24 September 2017

1

1 reference

https://scigraph.springernature.com/pub.10.1038/ki.1993.17

24 September 2017

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The mouse polycystic kidney disease mutation (cpk) is located on proximal chromosome 12

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Nephron obstruction in nordihydroguaiaretic acid-induced renal cystic disease

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Basement membrane alterations during development and regression of tubular cysts.

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Altered basement membrane protein biosynthesis by primary cultures of cpk/cpk mouse kidney

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A new method for studying human polycystic kidney disease epithelia in culture

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7 January 2021

Ischemia induces partial loss of surface membrane polarity and accumulation of putative calcium ionophores

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Ischemia induces surface membrane dysfunction. Mechanism of altered Na+-dependent glucose transport

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7 January 2021

Characterization of ischemia-induced loss of epithelial polarity

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In situ hybridization reveals temporal and spatial changes in cellular expression of mRNA for a laminin receptor, laminin, and basement membrane (type IV) collagen in the developing kidney

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7 January 2021

Identifiers

DOI

10.1038/KI.1993.17

1 reference

24 September 2017

1 reference

PubMed ID

8433548