(Q40877248)

English

The receptor for advanced glycation end products is induced by the glycation products themselves and tumor necrosis factor-alpha through nuclear factor-kappa B, and by 17beta-estradiol through Sp-1 in human vascular endothelial cells

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scholarly article

1 reference

Europe PubMed Central

24 September 2017

The receptor for advanced glycation end products is induced by the glycation products themselves and tumor necrosis factor-alpha through nuclear factor-kappa B, and by 17beta-estradiol through Sp-1 in human vascular endothelial cells (English)

1 reference

Europe PubMed Central

24 September 2017

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author name string

Tanaka N

1

1 reference

Europe PubMed Central

24 September 2017

Yonekura H

2

1 reference

Europe PubMed Central

24 September 2017

Yamagishi S

3

1 reference

Europe PubMed Central

24 September 2017

Fujimori H

4

1 reference

Europe PubMed Central

24 September 2017

Yamamoto Y

5

1 reference

Europe PubMed Central

24 September 2017

Yamamoto H

6

1 reference

Europe PubMed Central

24 September 2017

language of work or name

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publication date

1 August 2000

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Europe PubMed Central

24 September 2017

Journal of Biological Chemistry

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Europe PubMed Central

24 September 2017

275

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Europe PubMed Central

24 September 2017

25781-25790

1 reference

Europe PubMed Central

24 September 2017

33

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Europe PubMed Central

24 September 2017

Advanced glycosylation end products in tissue and the biochemical basis of diabetic complications

1 reference

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1 reference

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Activation of receptor for advanced glycation end products: a mechanism for chronic vascular dysfunction in diabetic vasculopathy and atherosclerosis

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Adipose expression of tumor necrosis factor-alpha: direct role in obesity-linked insulin resistance

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Tumor necrosis factor alpha: a key component of the obesity-diabetes link

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Increased adipose tissue expression of tumor necrosis factor-alpha in human obesity and insulin resistance

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An essential role for NF-kappaB in preventing TNF-alpha-induced cell death

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TNF- and cancer therapy-induced apoptosis: potentiation by inhibition of NF-kappaB

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Suppression of TNF-alpha-induced apoptosis by NF-kappaB

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Characterization and functional analysis of the promoter of RAGE, the receptor for advanced glycation end products

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A compilation of amino acid analyses of proteins. II.

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Three genes in the human MHC class III region near the junction with the class II: gene for receptor of advanced glycosylation end products, PBX2 homeobox gene and a notch homolog, human counterpart of mouse mammary tumor gene int-3

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Receptor for advanced glycation end products (AGEs) has a central role in vessel wall interactions and gene activation in response to circulating AGE proteins

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Advanced glycation endproducts interacting with their endothelial receptor induce expression of vascular cell adhesion molecule-1 (VCAM-1) in cultured human endothelial cells and in mice. A potential mechanism for the accelerated vasculopathy of dia

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Receptor-mediated endothelial cell dysfunction in diabetic vasculopathy. Soluble receptor for advanced glycation end products blocks hyperpermeability in diabetic rats

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Suppression of accelerated diabetic atherosclerosis by the soluble receptor for advanced glycation endproducts

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The effect of pregnancy on retinal hemodynamics in diabetic versus nondiabetic mothers

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An estrogen-responsive element derived from the 5' flanking region of the Xenopus vitellogenin A2 gene functions in transfected human cells

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Sp1-binding elements in the promoter of RAGE are essential for amphoterin-mediated gene expression in cultured neuroblastoma cells

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Estrogen-induced retinoic acid receptor alpha 1 gene expression: role of estrogen receptor-Sp1 complex

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Estrogen induces adenosine deaminase gene expression in MCF-7 human breast cancer cells: role of estrogen receptor-Sp1 interactions

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Transcriptional activation of insulin-like growth factor-binding protein-4 by 17beta-estradiol in MCF-7 cells: role of estrogen receptor-Sp1 complexes

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Identification by in vivo genomic footprinting of a transcriptional switch containing NF-kappaB and Sp1 that regulates the IkappaBalpha promoter

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TGFbeta signaling: receptors, transducers, and Mad proteins.

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Jak-STAT pathways and transcriptional activation in response to IFNs and other extracellular signaling proteins

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The receptor for advanced glycation end products (RAGE) is a cellular binding site for amphoterin. Mediation of neurite outgrowth and co-expression of rage and amphoterin in the developing nervous system

1 reference

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21 January 2018

RAGE mediates a novel proinflammatory axis: a central cell surface receptor for S100/calgranulin polypeptides

1 reference

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Advanced glycation end products increase retinal vascular endothelial growth factor expression

1 reference

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Transforming growth factor beta 1 down-regulates vascular endothelial growth factor receptor 2/flk-1 expression in vascular endothelial cells.

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Identifiers

10.1074/JBC.M001235200

1 reference

Europe PubMed Central

24 September 2017

1 reference

Europe PubMed Central

24 September 2017

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