(Q40890934)

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Suppression of malignant growth potentials of v-Src-transformed human gallbladder epithelial cells by adenovirus-mediated dominant negative H-Ras

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Europe PubMed Central

PubMed publication ID

24 September 2017

Suppression of malignant growth potentials of v-Src-transformed human gallbladder epithelial cells by adenovirus-mediated dominant negative H-Ras (English)

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

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author name string

Tokumitsu Y

1

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

Nakano S

2

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

Ueno H

3

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

Niho Y

4

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

publication date

1 May 2000

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

Journal of Cellular Physiology

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Europe PubMed Central

PubMed publication ID

24 September 2017

183

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

2

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

221-227

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

Ras-independent transformation by v-Src

1 reference

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Myc but not Fos rescue of PDGF signalling block caused by kinase-inactive Src.

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Activation of pp60c-src protein kinase activity in human colon carcinoma

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21 January 2018

Stat3 activation is required for cellular transformation by v-src

1 reference

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21 January 2018

Oncogenes and signal transduction

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21 January 2018

Activation and association of Stat3 with Src in v-Src-transformed cell lines

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Activation of the pp60c-src protein kinase is an early event in colonic carcinogenesis

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Suppression of src transformation by overexpression of full-length GTPase-activating protein (GAP) or of the GAP C terminus

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Inhibitors of anchorage-independent growth affect the growth of transformed cells on poly(2-hydroxyethyl methacrylate)-coated surfaces

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Signaling through focal adhesion kinase

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Activating SRC mutation in a subset of advanced human colon cancers

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Cell transformation by the viral src oncogene

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21 January 2018

Two types of RAS mutants that dominantly interfere with activators of RAS.

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21 January 2018

Activation of Rac1, RhoA, and mitogen-activated protein kinases is required for Ras transformation

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Tyrosine kinase inhibition: an approach to drug development

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Activation of c-Src by receptor tyrosine kinases in human colon cancer cells with high metastatic potential

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21 January 2018

Characterization of a newly established human gallbladder carcinoma cell line

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21 January 2018

Inhibition of v-src-induced transformation by a GTPase-activating protein

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21 January 2018

Ras, but not Src, transformation of RIE-1 epithelial cells is dependent on activation of the mitogen-activated protein kinase cascade

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Farnesyltransferase inhibition causes morphological reversion of ras-transformed cells by a complex mechanism that involves regulation of the actin cytoskeleton

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An essential role for Rac in Ras transformation

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A role for Rho in Ras transformation

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Cdc42 regulates anchorage-independent growth and is necessary for Ras transformation

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Requirement for c-ras proteins during viral oncogene transformation

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Direct tumorigenic conversion of human gallbladder carcinoma cells by v-src but not by activated c-H-ras oncogene.

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Quantitative analysis of repeat adenovirus-mediated gene transfer into injured canine femoral arteries

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Identifiers

10.1002/(SICI)1097-4652(200005)183:2<221::AID-JCP8>3.0.CO;2-L

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

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