(Q40894283)

24 September 2017

title

Apoptotic response to growth factor deprivation involves cooperative interactions between c-Fos and p300. (English)

1 reference

24 September 2017

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Preston GA

1

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24 September 2017

Srinivasan D

series ordinal

2

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24 September 2017

Barrett JC

series ordinal

3

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24 September 2017

publication date

1 February 2000

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Cell Death & Differentiation

24 September 2017

published in

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24 September 2017

volume

7

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24 September 2017

issue

2

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24 September 2017

page(s)

215-226

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https://scigraph.springernature.com/pub.10.1038/sj.cdd.4400637

24 September 2017

exact match

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cites work

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The proto-oncogene c-fos increases the sensitivity of keratinocytes to apoptosis

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Treatment of melanoma cells with the synthetic retinoid CD437 induces apoptosis via activation of AP-1 in vitro, and causes growth inhibition in xenografts in vivo

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An essential role for NF-kappaB in preventing TNF-alpha-induced cell death

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Functional interplay between p53 and E2F through co-activator p300.

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Role of EGR-1 in thapsigargin-inducible apoptosis in the melanoma cell line A375-C6.

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Resistance to apoptosis in CTLL-2 cells constitutively expressing c-Myb is associated with induction of BCL-2 expression and Myb-dependent regulation of bcl-2 promoter activity

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p53-dependent apoptosis in the absence of transcriptional activation of p53-target genes

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Mediation of c-Myc-induced apoptosis by p53.

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Retinoid-induced apoptosis and Sp1 cleavage occur independently of transcription and require caspase activation

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Cell death: the significance of apoptosis

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An essential role for p300/CBP in the cellular response to hypoxia

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Differential transcriptional activation by human T-cell leukemia virus type 1 Tax mutants is mediated by distinct interactions with CREB binding protein and p300

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Cooperation of Stat2 and p300/CBP in signalling induced by interferon-alpha

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Regulatory domains of the A-Myb transcription factor and its interaction with the CBP/p300 adaptor molecules

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CREB-binding protein/p300 are transcriptional coactivators of p65

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The Adenovirus E1A-associated 300-kD Protein Exhibits Properties of a Transcriptional Coactivator and Belongs to an Evolutionarily Conserved Family

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Molecular cloning and functional analysis of the adenovirus E1A-associated 300-kD protein (p300) reveals a protein with properties of a transcriptional adaptor

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The adenovirus E1A proteins induce apoptosis, which is inhibited by the E1B 19-kDa and Bcl-2 proteins

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Role of a tumor-suppressor gene in the negative control of anchorage-independent growth of Syrian hamster cells

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Identification of genes associated with tumor suppression in Syrian hamster embryo cells

21 January 2018

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Adenovirus E1A downregulates cJun- and JunB-mediated transcription by targeting their coactivator p300

21 January 2018

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Binding and modulation of p53 by p300/CBP coactivators

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Recruitment of p300/CBP in p53-dependent signal pathways

21 January 2018

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Relief of YY1 transcriptional repression by adenovirus E1A is mediated by E1A-associated protein p300

21 January 2018

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p300 and CBP: partners for life and death

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High levels of c-rel expression are associated with programmed cell death in the developing avian embryo and in bone marrow cells in vitro

21 January 2018

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7 January 2021

Selective c-Jun overexpression is associated with ionizing radiation-induced apoptosis in the developing cerebellum of the rat

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7 January 2021

Strong c-Jun immunoreactivity is associated with apoptotic cell death in human tumors of the central nervous system

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7 January 2021

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7 January 2021

Role for p300 in stabilization of p53 in the response to DNA damage

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7 January 2021

Identifiers

DOI

10.1038/SJ.CDD.4400637

1 reference

Dimensions Publication ID

24 September 2017

1023347163

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1 reference