(Q40916192)

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Bax:Bcl-2 ratio modulation by bryostatin 1 and novel antitubulin agents is important for susceptibility to drug induced apoptosis in the human early pre-B acute lymphoblastic leukemia cell line, Reh.

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Europe PubMed Central

PubMed publication ID

24 September 2017

Bax:Bcl-2 ratio modulation by bryostatin 1 and novel antitubulin agents is important for susceptibility to drug induced apoptosis in the human early pre-B acute lymphoblastic leukemia cell line, Reh. (English)

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Europe PubMed Central

PubMed publication ID

24 September 2017

lymphoblastic leukemia

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1

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24 September 2017

author name string

Mohammad RM

2

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24 September 2017

Al-Katib AM

3

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Europe PubMed Central

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24 September 2017

publication date

1 October 1999

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Europe PubMed Central

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24 September 2017

Leukemia Research

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Europe PubMed Central

PubMed publication ID

24 September 2017

23

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PubMed publication ID

24 September 2017

10

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Europe PubMed Central

PubMed publication ID

24 September 2017

881-888

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Europe PubMed Central

PubMed publication ID

24 September 2017

Molecular and clinical aspects of apoptosis

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Apoptosis: A Basic Biological Phenomenon with Wideranging Implications in Tissue Kinetics

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Social controls on cell survival and cell death

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Mechanisms of apoptotic cell death

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Agents that down-regulate or inhibit protein kinase C circumvent resistance to 1-beta-D-arabinofuranosylcytosine-induced apoptosis in human leukemia cells that overexpress Bcl-2.

1 reference

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Drug-target interactions: only the first step in the commitment to a programmed cell death?

1 reference

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p53-dependent apoptosis modulates the cytotoxicity of anticancer agents

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Bcl-2 gene promotes haemopoietic cell survival and cooperates with c-myc to immortalize pre-B cells

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Therapeutics: a glimpse of the future

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In vitro cytotoxicity of docetaxel in childhood acute leukemias

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Mechanism of UV-induced apoptosis in human leukemia cells: roles of Ca2+/Mg(2+)-dependent endonuclease, caspase-3, and stress-activated protein kinases

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Cellular oxidative stress and the control of apoptosis by wild-type p53, cytotoxic compounds, and cytokines

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Evidence for the involvement of endothelial cell integrin alphaVbeta3 in the disruption of the tumor vasculature induced by TNF and IFN-gamma

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TGF-beta inhibits growth and induces apoptosis in leukemic B cell precursors

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In vitro down-regulation of bcl-2 expression by all-trans retinoic acid in AML blasts

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Modulation of p53, WAF1/p21 and BCL-2 expression during retinoic acid-induced differentiation of NB4 promyelocytic cells

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Localization of granzyme B in the nucleus. A putative role in the mechanism of cytotoxic lymphocyte-mediated apoptosis

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Differential involvement of caspases in apoptosis of myeloid leukemic cells induced by chemotherapy versus growth factor withdrawal

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Implication of mitochondrial hydrogen peroxide generation in ceramide-induced apoptosis

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High expression of bcl-2 is the rule in acute lymphoblastic leukemia, except in Burkitt subtype at presentation, and is not correlated with the prognosis

1 reference

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The Bax alpha:Bcl-2 ratio modulates the response to dexamethasone in leukaemic cells and is highly variable in childhood acute leukaemia

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Surface immunoglobulin light-chain expression by the "common" all cell line REH.

1 reference

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Isolation and structure of bryostatin 1

1 reference

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Comparative effects of polymyxin B, phorbol ester and bryostatin on protein phosphorylation, protein kinase C translocation, phospholipid metabolism and differentiation of HL60 cells

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Differential effects of active isomers, segments, and analogs of dolastatin 10 on ligand interactions with tubulin. Correlation with cytotoxicity

1 reference

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The bcl-2 and p53 oncoproteins can be modulated by bryostatin 1 and dolastatins in human diffuse large cell lymphoma

1 reference

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Synergistic interaction of selected marine animal anticancer drugs against human diffuse large cell lymphoma

1 reference

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Bcl-2 phosphorylation required for anti-apoptosis function

1 reference

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Reversible phosphorylation of Bcl2 following interleukin 3 or bryostatin 1 is mediated by direct interaction with protein phosphatase 2A

1 reference

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Reversal of chemoresistance of lymphoma cells by antisense-mediated reduction of bcl-2 gene expression.

1 reference

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Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programmed cell death

1 reference

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BH1 and BH2 domains of Bcl-2 are required for inhibition of apoptosis and heterodimerization with Bax.

1 reference

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Identifiers

10.1016/S0145-2126(99)00108-3

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

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