(Q40918991)

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The alpha-isoform of class II phosphoinositide 3-kinase is more effectively activated by insulin receptors than IGF receptors, and activation requires receptor NPEY motifs

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scholarly article

1 reference

Europe PubMed Central

24 September 2017

The alpha-isoform of class II phosphoinositide 3-kinase is more effectively activated by insulin receptors than IGF receptors, and activation requires receptor NPEY motifs (English)

1 reference

Europe PubMed Central

24 September 2017

author name string

Ursø B

1

1 reference

Europe PubMed Central

24 September 2017

Brown RA

2

1 reference

Europe PubMed Central

24 September 2017

O'Rahilly S

3

1 reference

Europe PubMed Central

24 September 2017

Shepherd PR

4

1 reference

Europe PubMed Central

24 September 2017

Siddle K

5

1 reference

Europe PubMed Central

24 September 2017

language of work or name

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publication date

1 November 1999

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Europe PubMed Central

24 September 2017

1 reference

Europe PubMed Central

24 September 2017

460

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Europe PubMed Central

24 September 2017

423-426

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Europe PubMed Central

24 September 2017

3

1 reference

Europe PubMed Central

24 September 2017

Phosphoinositide 3-kinase: the key switch mechanism in insulin signalling

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2899%2901388-5

21 January 2018

Phosphoinositide 3-kinases: a conserved family of signal transducers

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2899%2901388-5

21 January 2018

Insulin activates the alpha isoform of class II phosphoinositide 3-kinase

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2899%2901388-5

21 January 2018

The signaling potential of the receptors for insulin and insulin-like growth factor I (IGF-I) in 3T3-L1 adipocytes: comparison of glucose transport activity, induction of oncogene c-fos, glucose transporter mRNA, and DNA-synthesis

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2899%2901388-5

21 January 2018

Evidence for a differential interaction of SHC and the insulin receptor substrate-1 (IRS-1) with the insulin-like growth factor-I (IGF-I) receptor in the yeast two-hybrid system

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2899%2901388-5

21 January 2018

Phosphotyrosine-dependent interaction of SHC and insulin receptor substrate 1 with the NPEY motif of the insulin receptor via a novel non-SH2 domain

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2899%2901388-5

21 January 2018

Non-SH2 domains within insulin receptor substrate-1 and SHC mediate their phosphotyrosine-dependent interaction with the NPEY motif of the insulin-like growth factor I receptor

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2899%2901388-5

21 January 2018

Differential signaling to glycogen synthesis by the intracellular domain of the insulin versus the insulin-like growth factor-1 receptor. Evidence from studies of TrkC-chimeras

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2899%2901388-5

21 January 2018

A novel class II phosphoinositide 3-kinase predominantly expressed in the liver and its enhanced expression during liver regeneration

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2899%2901388-5

21 January 2018

The insulin signalling system and the IRS proteins

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2899%2901388-5

21 January 2018

PTB domains of IRS-1 and Shc have distinct but overlapping binding specificities

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2899%2901388-5

21 January 2018

Distinct modes of interaction of SHC and insulin receptor substrate-1 with the insulin receptor NPEY region via non-SH2 domains

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2899%2901388-5

21 January 2018

The role of the NPXY motif in the insulin receptor in tyrosine phosphorylation of insulin receptor substrate-1 and Shc

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2899%2901388-5

21 January 2018

Identifiers

10.1016/S0014-5793(99)01388-5

1 reference

Europe PubMed Central

24 September 2017

1 reference

Europe PubMed Central

24 September 2017

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