(Q41017727)

25 September 2017

Overproduction of the CFTR R domain leads to increased levels of asialoGM1 and increased Pseudomonas aeruginosa binding by epithelial cells (English)

1 reference

25 September 2017

Pseudomonas aeruginosa

0 references

author name string

Bryan R

1

1 reference

25 September 2017

Kube D

2

1 reference

25 September 2017

Perez A

3

1 reference

25 September 2017

Davis P

4

1 reference

25 September 2017

Prince A

5

1 reference

25 September 2017

language of work or name

1 reference

1 August 1998

1 reference

American Journal of Respiratory Cell and Molecular Biology

25 September 2017

1 reference

25 September 2017

19

1 reference

25 September 2017

269-277

1 reference

25 September 2017

2

1 reference

Cystic fibrosis airway epithelia fail to kill bacteria because of abnormal airway surface fluid.

25 September 2017

1 reference

https://api.crossref.org/works/10.1165%2FAJRCMB.19.2.2889

Pseudomonas aeruginosa pili bind to asialoGM1 which is increased on the surface of cystic fibrosis epithelial cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1165%2FAJRCMB.19.2.2889

Cystic fibrosis epithelial cells have a receptor for pathogenic bacteria on their apical surface

21 January 2018

1 reference

https://api.crossref.org/works/10.1165%2FAJRCMB.19.2.2889

Diverse Pseudomonas aeruginosa gene products stimulate respiratory epithelial cells to produce interleukin-8

21 January 2018

1 reference

https://api.crossref.org/works/10.1165%2FAJRCMB.19.2.2889

Modulation of airway inflammation in cystic fibrosis. In vivo suppression of interleukin-8 levels on the respiratory epithelial surface by aerosolization of recombinant secretory leukoprotease inhibitor

21 January 2018

1 reference

https://api.crossref.org/works/10.1165%2FAJRCMB.19.2.2889

Inflammatory cytokines in cystic fibrosis lungs

21 January 2018

1 reference

https://api.crossref.org/works/10.1165%2FAJRCMB.19.2.2889

Defective acidification of intracellular organelles in cystic fibrosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1165%2FAJRCMB.19.2.2889

Increased sulfation of glycoconjugates by cultured nasal epithelial cells from patients with cystic fibrosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1165%2FAJRCMB.19.2.2889

Evidence against defective trans-Golgi acidification in cystic fibrosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1165%2FAJRCMB.19.2.2889

Molecular mechanisms of CFTR chloride channel dysfunction in cystic fibrosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1165%2FAJRCMB.19.2.2889

Phosphorylation of the R domain by cAMP-dependent protein kinase regulates the CFTR chloride channel

21 January 2018

1 reference

https://api.crossref.org/works/10.1165%2FAJRCMB.19.2.2889

Phosphorylation-dependent block of cystic fibrosis transmembrane conductance regulator chloride channel by exogenous R domain protein

21 January 2018

1 reference

https://api.crossref.org/works/10.1165%2FAJRCMB.19.2.2889

Phosphorylation by cAMP-dependent protein kinase causes a conformational change in the R domain of the cystic fibrosis transmembrane conductance regulator

21 January 2018

1 reference

https://api.crossref.org/works/10.1165%2FAJRCMB.19.2.2889

Characterization of human tracheal epithelial cells transformed by an origin-defective simian virus 40.

21 January 2018

1 reference

https://api.crossref.org/works/10.1165%2FAJRCMB.19.2.2889

Activation of transcription factor NF-kappaB by the adenovirus E3/19K protein requires its ER retention

21 January 2018

1 reference

https://api.crossref.org/works/10.1165%2FAJRCMB.19.2.2889

Role of mutant CFTR in hypersusceptibility of cystic fibrosis patients to lung infections.

21 January 2018

1 reference

https://api.crossref.org/works/10.1165%2FAJRCMB.19.2.2889

21 January 2018

Identifiers

DOI

10.1165/AJRCMB.19.2.2889

1 reference

25 September 2017

1 reference