(Q41134815)

English

Chronic eticlopride and dopamine denervation induce equal nonadditive increases in striatal D2 receptor density: Autoradiographic evidence against the dual mechanism hypothesis

scientific article published on January 1, 1991

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Europe PubMed Central

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27 September 2017

Chronic eticlopride and dopamine denervation induce equal nonadditive increases in striatal D2 receptor density: Autoradiographic evidence against the dual mechanism hypothesis (English)

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10.1016/0306-4522(91)90342-L

https://api.crossref.org/works/10.1016/0306-4522(91)90342-L

27 October 2022

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LaHoste GJ

1

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Europe PubMed Central

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27 September 2017

G. J. LaHoste

1

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Europe PubMed Central

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?resulttype=core&format=json&query=SRC%3AMED%20AND%20EXT_ID%3A1678501

27 October 2022

J. F. Marshall

2

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Europe PubMed Central

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?resulttype=core&format=json&query=SRC%3AMED%20AND%20EXT_ID%3A1678501

27 October 2022

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1 January 1991

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27 September 2017

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https://api.elsevier.com/content/article/PII:030645229190342L?httpAccept=text/xml

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10.1016/0306-4522(91)90342-L

https://api.crossref.org/works/10.1016/0306-4522(91)90342-L

27 October 2022

https://api.elsevier.com/content/article/PII:030645229190342L?httpAccept=text/plain

1 reference

10.1016/0306-4522(91)90342-L

https://api.crossref.org/works/10.1016/0306-4522(91)90342-L

27 October 2022

1 reference

Europe PubMed Central

PubMed publication ID

27 September 2017

41

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PubMed publication ID

27 September 2017

2-3

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Europe PubMed Central

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27 September 2017

473-481

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Europe PubMed Central

PubMed publication ID

27 September 2017

Time course of adaptations in dopamine biosynthesis, metabolism, and release following nigrostriatal lesions: implications for behavioral recovery from brain injury

1

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Brain dopamine and serotonin receptor sites revealed by digital subtraction autoradiography

2

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Computer-assisted video analysis of [3H]spiroperidol binding autoradiographs.

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Efferent connections of the substantia nigra and ventral tegmental area in the rat

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Cognitive Deficit Caused by Regional Depletion of Dopamine in Prefrontal Cortex of Rhesus Monkey

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Acute and chronic haloperidol treatment: Comparison of effects on nigral dopaminergic cell activity

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Antischizophrenic drugs: chronic treatment elevates dopamine receptor binding in brain

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Nigrostriatal lesions enhance striatal 3H-apomorphine and 3H-spiroperidol binding

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The effects of dose and duration of chronic pimozide administration on dopamine receptor supersensitivity

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Catecholamine innervation of the basal forebrain. IV. Topography of the dopamine projection to the basal forebrain and neostriatum

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Some in vitro receptor binding properties of [3H]eticlopride, a novel substituted benzamide, selective for dopamine-D2 receptors in the rat brain

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Chronic treatment with dopamine receptor antagonists: behavioral and pharmacologic effects on D1 and D2 dopamine receptors

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A behavioural and biochemical comparison of dopamine receptor blockade produced by haloperidol with that produced by substituted benzamide drugs

13

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Dopamine D-2 receptors in rat caudate-putamen: the lateral to medial gradient does not correspond to dopaminergic innervation

14

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Multiple receptors for dopamine

15

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Non-additivity of D2 receptor proliferation induced by dopamine denervation and chronic selective antagonist administration: evidence from quantitative autoradiography indicates a single mechanism of action

17

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Nigral D1 and striatal D2 receptors mediate the behavioral effects of dopamine agonists

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Quantification of lesion-induced dopaminergic supersensitivity using the rotational model in the mouse

19

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Somatosensory inattention after dopamine-depleting intracerebral 6-OHDA injections: Spontaneous recovery and pharmacological control

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Brain Function: Neural Adaptations and Recovery From Injury

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Supersensitivity to apomorphine following destruction of the ascending dopamine neurons: Quantification using the rotational model

22

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Decreased striatal D1 binding density following mesotelencephalic 6-hydroxydopamine injections: an autoradiographic analysis

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Effects of chronic treatment with selective and nonselective antagonists on the subtypes of dopamine receptors

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Plasticity of neostriatal dopamine receptors after nigrostriatal injury: relationship to recovery of sensorimotor functions and behavioral supersensitivity.

26

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The effects of denervation and chronic haloperidol treatment on neostriatal dopamine receptor density are not additive in the rat.

27

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Chronic treatment with clozapine or haloperidol differentially regulates dopamine and serotonin receptors in rat brain

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Quantification of dopaminergic supersensitization using apomorphine-induced behavior in the mouse

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Dopamine receptors in the denervated striatum: further supersensitivity by chronic haloperidol treatment

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CNS dopamine autoreceptors: distribution, pharmacology, and function

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Chronic treatment with clozapine, unlike haloperidol, does not induce changes in striatal D-2 receptor function in the rat.

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Different plasticity changes in D1 and D2 receptors in rat striatal subregions following impairment of dopaminergic transmission

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Denervation supersensitivity of striatal D2 dopamine receptors is restricted to the ventro- and dorsolateral regions of the striatum

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Autoradiographic localization of D1 dopamine receptors in the rat brain with [3H]SCH 23390.

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Dopaminergic A10 neurones are involved in cognitive functions.

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Dopaminergic supersensitivity induced by denervation and chronic receptor blockade is additive

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Dopamine receptor changes following destruction of the nigrostriatal pathway: Lack of a relationship to rotational behavior

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Reserpine-induced up-regulation of dopamine D2 receptors in the rat striatum is enhanced by denervation but not by chronic receptor blockade

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Comparison of the effects of chronic haloperidol treatment on A9 and A10 dopamine neurons in the rat

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Neurochemical Compensation After Nigrostriatal Bundle Injury in an Animal Model of Preclinical Parkinsonism

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27 October 2022

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10.1016/0306-4522(91)90342-L

1 reference

Europe PubMed Central

PubMed publication ID

27 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

27 September 2017

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