(Q41197271)

English

The endoplasmic reticulum degradation pathway for mutant secretory proteins alpha1-antitrypsin Z and S is distinct from that for an unassembled membrane protein

scientific article published on May 1996

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

28 September 2017

The endoplasmic reticulum degradation pathway for mutant secretory proteins alpha1-antitrypsin Z and S is distinct from that for an unassembled membrane protein (English)

1 reference

Europe PubMed Central

PubMed publication ID

28 September 2017

endoplasmic reticulum

0 references

membrane protein

1 reference

based on heuristic

inferred from title

author name string

Teckman JH

1

1 reference

Europe PubMed Central

PubMed publication ID

28 September 2017

Perlmutter DH

2

1 reference

Europe PubMed Central

PubMed publication ID

28 September 2017

language of work or name

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publication date

1 May 1996

1 reference

Europe PubMed Central

PubMed publication ID

28 September 2017

Journal of Biological Chemistry

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Europe PubMed Central

PubMed publication ID

28 September 2017

271

1 reference

Europe PubMed Central

PubMed publication ID

28 September 2017

22

1 reference

Europe PubMed Central

PubMed publication ID

28 September 2017

13215-13220

1 reference

Europe PubMed Central

PubMed publication ID

28 September 2017

Alpha 1-antitrypsin deficiency, emphysema, and liver disease. Genetic basis and strategies for therapy

1 reference

https://api.crossref.org/works/10.1074%2FJBC.271.22.13215

21 January 2018

Accumulation of PiZ alpha 1-antitrypsin causes liver damage in transgenic mice

1 reference

https://api.crossref.org/works/10.1074%2FJBC.271.22.13215

21 January 2018

A lag in intracellular degradation of mutant alpha 1-antitrypsin correlates with the liver disease phenotype in homozygous PiZZ alpha 1-antitrypsin deficiency

1 reference

https://api.crossref.org/works/10.1074%2FJBC.271.22.13215

21 January 2018

Protein degradation in the endoplasmic reticulum

1 reference

https://api.crossref.org/works/10.1074%2FJBC.271.22.13215

21 January 2018

The two subunits of the human asialoglycoprotein receptor have different fates when expressed alone in fibroblasts

1 reference

https://api.crossref.org/works/10.1074%2FJBC.271.22.13215

21 January 2018

Inhibition of restriction endonuclease Nci I cleavage by phosphorothioate groups and its application to oligonucleotide-directed mutagenesis

1 reference

https://api.crossref.org/works/10.1074%2FJBC.271.22.13215

21 January 2018

Site-directed mutagenesis by overlap extension using the polymerase chain reaction

1 reference

https://api.crossref.org/works/10.1074%2FJBC.271.22.13215

21 January 2018

Expression of a foreign gene in myeloid and lymphoid cells derived from multipotent haematopoietic precursors.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.271.22.13215

21 January 2018

Redesign of retrovirus packaging cell lines to avoid recombination leading to helper virus production

1 reference

https://api.crossref.org/works/10.1074%2FJBC.271.22.13215

21 January 2018

Dissociation of a 110-kD peripheral membrane protein from the Golgi apparatus is an early event in brefeldin A action

1 reference

https://api.crossref.org/works/10.1074%2FJBC.271.22.13215

21 January 2018

PtK1 cells contain a nondiffusible, dominant factor that makes the Golgi apparatus resistant to brefeldin A

1 reference

https://api.crossref.org/works/10.1074%2FJBC.271.22.13215

21 January 2018

Two pathways for the degradation of the H2 subunit of the asialoglycoprotein receptor in the endoplasmic reticulum

1 reference

https://api.crossref.org/works/10.1074%2FJBC.271.22.13215

21 January 2018

Carboxy terminally truncated forms of ribophorin I are degraded in pre-Golgi compartments by a calcium-dependent process

1 reference

https://api.crossref.org/works/10.1074%2FJBC.271.22.13215

21 January 2018

Defective intracellular transport and processing of CFTR is the molecular basis of most cystic fibrosis

1 reference

https://api.crossref.org/works/10.1074%2FJBC.271.22.13215

21 January 2018

Degradation of CFTR by the ubiquitin-proteasome pathway

1 reference

https://api.crossref.org/works/10.1074%2FJBC.271.22.13215

21 January 2018

Multiple proteolytic systems, including the proteasome, contribute to CFTR processing.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.271.22.13215

21 January 2018

Identifiers

10.1074/JBC.271.22.13215

1 reference

Europe PubMed Central

PubMed publication ID

28 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

28 September 2017

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