(Q41319987)

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Wnt11 gene therapy with adeno-associated virus 9 improves the survival of mice with myocarditis induced by coxsackievirus B3 through the suppression of the inflammatory reaction

scientific article published on 14 April 2015

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Europe PubMed Central

PubMed publication ID

29 September 2017

Wnt11 gene therapy with adeno-associated virus 9 improves the survival of mice with myocarditis induced by coxsackievirus B3 through the suppression of the inflammatory reaction (English)

1 reference

Europe PubMed Central

PubMed publication ID

29 September 2017

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author name string

Yutaka Aoyama

1

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Europe PubMed Central

PubMed publication ID

29 September 2017

Koichi Kobayashi

2

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Europe PubMed Central

PubMed publication ID

29 September 2017

Yoshihiro Morishita

3

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Europe PubMed Central

PubMed publication ID

29 September 2017

Kengo Maeda

4

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Europe PubMed Central

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29 September 2017

Toyoaki Murohara

5

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Europe PubMed Central

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29 September 2017

publication date

14 April 2015

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Europe PubMed Central

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29 September 2017

Journal of Molecular and Cellular Cardiology

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Europe PubMed Central

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29 September 2017

84

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Europe PubMed Central

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29 September 2017

45-51

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Europe PubMed Central

PubMed publication ID

29 September 2017

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.04.009

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Coxsackie B myopericarditis in adults

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Underlying causes and long-term survival in patients with initially unexplained cardiomyopathy

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Fulminant myocarditis

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Advances in the understanding of myocarditis

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Long-term outcome of fulminant myocarditis as compared with acute (nonfulminant) myocarditis

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From myocarditis to cardiomyopathy: mechanisms of inflammation and cell death: learning from the past for the future

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Coxsackievirus B3 induces T regulatory cells, which inhibit cardiomyopathy in tumor necrosis factor-alpha transgenic mice

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Remission of CVB3-induced viral myocarditis by in vivo Th2 polarization via hydrodynamics-based interleukin-4 gene transfer

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A clinical trial of immunosuppressive therapy for myocarditis. The Myocarditis Treatment Trial Investigators

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Wnt/beta-catenin signaling: components, mechanisms, and diseases

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The Wingless homolog WNT5A and its receptor Frizzled-5 regulate inflammatory responses of human mononuclear cells induced by microbial stimulation

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Wnt5A/CaMKII signaling contributes to the inflammatory response of macrophages and is a target for the antiinflammatory action of activated protein C and interleukin-10

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Wnt-11 activation of a non-canonical Wnt signalling pathway is required for cardiogenesis

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Wingless homolog Wnt11 suppresses bacterial invasion and inflammation in intestinal epithelial cells

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Wnt11 and Ret/Gdnf pathways cooperate in regulating ureteric branching during metanephric kidney development.

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7 January 2021

Effects of in vivo administration of anti-B7-1/B7-2 monoclonal antibodies on murine acute myocarditis caused by coxsackievirus B3

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Robust systemic transduction with AAV9 vectors in mice: efficient global cardiac gene transfer superior to that of AAV8.

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Cardiac troponin I but not cardiac troponin T induces severe autoimmune inflammation in the myocardium

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Secreted Frizzled-related protein 2 is a procollagen C proteinase enhancer with a role in fibrosis associated with myocardial infarction

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CXCL10 inhibits viral replication through recruitment of natural killer cells in coxsackievirus B3-induced myocarditis

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Innate defense mechanism against virus infection within the cardiac myocyte requiring gp130-STAT3 signaling

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The suppressor of cytokine signaling-1 (SOCS1) is a novel therapeutic target for enterovirus-induced cardiac injury

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Immunopathological basis of virus-induced myocarditis

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Update on myocarditis

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10.1016/J.YJMCC.2015.04.009

1 reference

Europe PubMed Central

PubMed publication ID

29 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

29 September 2017

ResearchGate publication ID

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