(Q41321495)

29 September 2017

title

Regulation of cell surface APO-1/Fas (CD95) ligand expression by metalloproteases (English)

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29 September 2017

Mariani SM

1

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29 September 2017

Matiba B

series ordinal

2

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29 September 2017

Bäumler C

series ordinal

3

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29 September 2017

Krammer PH

series ordinal

4

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29 September 2017

language of work or name

English

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publication date

1 August 1995

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European Journal of Immunology

29 September 2017

published in

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29 September 2017

volume

25

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29 September 2017

issue

8

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29 September 2017

page(s)

2303-2307

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The role of APO-1-mediated apoptosis in the immune system

29 September 2017

cites work

1 reference

https://api.crossref.org/works/10.1002%2FEJI.1830250828

7 January 2021

https://api.crossref.org/works/10.1002%2FEJI.1830250828

1 reference

7 January 2021

Lymphoproliferation disorder in mice explained by defects in Fas antigen that mediates apoptosis

1 reference

https://api.crossref.org/works/10.1002%2FEJI.1830250828

7 January 2021

Molecular cloning and expression of the Fas ligand, a novel member of the tumor necrosis factor family

1 reference

https://api.crossref.org/works/10.1002%2FEJI.1830250828

7 January 2021

Purification and characterization of the Fas-ligand that induces apoptosis

1 reference

https://api.crossref.org/works/10.1002%2FEJI.1830250828

7 January 2021

Generalized lymphoproliferative disease in mice, caused by a point mutation in the Fas ligand

1 reference

https://api.crossref.org/works/10.1002%2FEJI.1830250828

7 January 2021

The mouse Fas-ligand gene is mutated in gld mice and is part of a TNF family gene cluster

1 reference

https://api.crossref.org/works/10.1002%2FEJI.1830250828

7 January 2021

Autocrine T-cell suicide mediated by APO-1/(Fas/CD95)

1 reference

https://api.crossref.org/works/10.1002%2FEJI.1830250828

7 January 2021

Cell-autonomous Fas (CD95)/Fas-ligand interaction mediates activation-induced apoptosis in T-cell hybridomas.

1 reference

https://api.crossref.org/works/10.1002%2FEJI.1830250828

7 January 2021

Fas(CD95)/FasL interactions required for programmed cell death after T-cell activation.

1 reference

https://api.crossref.org/works/10.1002%2FEJI.1830250828

7 January 2021

Fas and perforin pathways as major mechanisms of T cell-mediated cytotoxicity

1 reference

https://api.crossref.org/works/10.1002%2FEJI.1830250828

7 January 2021

Expression of the functional soluble form of human fas ligand in activated lymphocytes.

1 reference

https://api.crossref.org/works/10.1002%2FEJI.1830250828

7 January 2021

The APO-1/Fas (CD95) receptor is expressed in homozygous MRL/lpr mice

1 reference

https://api.crossref.org/works/10.1002%2FEJI.1830250828

7 January 2021

Protection against a lethal dose of endotoxin by an inhibitor of tumour necrosis factor processing

1 reference

https://api.crossref.org/works/10.1002%2FEJI.1830250828

7 January 2021

Processing of tumour necrosis factor-α precursor by metalloproteinases

1 reference

https://api.crossref.org/works/10.1002%2FEJI.1830250828

7 January 2021

Regulation of tumour necrosis factor-alpha processing by a metalloproteinase inhibitor

1 reference

https://api.crossref.org/works/10.1002%2FEJI.1830250828

7 January 2021

A metalloprotease inhibitor blocks shedding of the 80-kD TNF receptor and TNF processing in T lymphocytes

1 reference

https://api.crossref.org/works/10.1002%2FEJI.1830250828

7 January 2021

A novel form of TNF/cachectin is a cell surface cytotoxic transmembrane protein: ramifications for the complex physiology of TNF

1 reference

https://api.crossref.org/works/10.1002%2FEJI.1830250828

7 January 2021

Identifiers

DOI

10.1002/EJI.1830250828

1 reference

29 September 2017

1 reference