(Q41325266)

29 September 2017

Analysis of the complement C3 fragments associated with hemodialysis membranes (English)

1 reference

29 September 2017

Cheung AK

1

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29 September 2017

Parker CJ

series ordinal

2

1 reference

29 September 2017

Janatova J

series ordinal

3

1 reference

29 September 2017

language of work or name

English

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publication date

1 February 1989

1 reference

29 September 2017

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29 September 2017

35

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29 September 2017

2

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29 September 2017

576-588

1 reference

https://scigraph.springernature.com/pub.10.1038/ki.1989.26

29 September 2017

0 references

cites work

Hemodialysis leukopenia. Pulmonary vascular leukostasis resulting from complement activation by dialyzer cellophane membranes

1 reference

https://api.crossref.org/works/10.1038%2FKI.1989.26

7 January 2021

Anaphylatoxin formation during hemodialysis: effects of different dialyzer membranes

1 reference

https://api.crossref.org/works/10.1038%2FKI.1989.26

7 January 2021

Complement and leukocyte-mediated pulmonary dysfunction in hemodialysis

1 reference

https://api.crossref.org/works/10.1038%2FKI.1989.26

7 January 2021

Anaphylatoxin formation during hemodialysis: comparison of new and re-used dialyzers

1 reference

https://api.crossref.org/works/10.1038%2FKI.1989.26

7 January 2021

Biocompatibility of dialysis membranes: Effects of chronic complement activation

1 reference

https://api.crossref.org/works/10.1038%2FKI.1989.26

7 January 2021

Acute cardiopulmonary decompensation and complement activation during hemodialysis

1 reference

https://api.crossref.org/works/10.1038%2FKI.1989.26

7 January 2021

Cardiopulmonary effects of cuprophane-activated plasma in the swine

1 reference

https://api.crossref.org/works/10.1038%2FKI.1989.26

7 January 2021

Complement Activation and Hypersensitivity Reactions to Dialysis Membranes

1 reference

https://api.crossref.org/works/10.1038%2FKI.1989.26

7 January 2021

Current concepts in immunology: the alternative pathway of complement--a system for host resistance to microbial infection.

1 reference

https://api.crossref.org/works/10.1038%2FKI.1989.26

7 January 2021

Relation of putative thioester bond in C3 to activation of the alternative pathway and the binding of C3b to biological targets of complement

1 reference

https://api.crossref.org/works/10.1038%2FKI.1989.26

7 January 2021

Evidence for presence of an internal thiolester bond in third component of human complement.

1 reference

https://api.crossref.org/works/10.1038%2FKI.1989.26

7 January 2021

Third component of human complement: Localization of the internal thiolester bond

1 reference

https://api.crossref.org/works/10.1038%2FKI.1989.26

7 January 2021

Binding reaction between the third human complement protein and small molecules

1 reference

https://api.crossref.org/works/10.1038%2FKI.1989.26

7 January 2021

Interaction between the third complement protein and cell surface macromolecules

1 reference

https://api.crossref.org/works/10.1038%2FKI.1989.26

7 January 2021

Covalent binding and hemolytic activity of complement proteins

1 reference

https://api.crossref.org/works/10.1038%2FKI.1989.26

7 January 2021

Effect of dialyzer reprocessing methods on complement activation and hemodialyzer-related symptoms

1 reference

https://api.crossref.org/works/10.1038%2FKI.1989.26

7 January 2021

Hemodialysis neutropenia and dialyzer reuse: role of the cleansing agent

1 reference

https://api.crossref.org/works/10.1038%2FKI.1989.26

7 January 2021

Symptoms and activation of granulocytes and complement with two dialysis membranes

1 reference

https://api.crossref.org/works/10.1038%2FKI.1989.26

7 January 2021

Compartmental distribution of complement activation products in artificial kidneys

1 reference

https://api.crossref.org/works/10.1038%2FKI.1989.26

7 January 2021

Complement Activation During Hemodialysis: Clinical Observations, Proposed Mechanisms, and Theoretical Implications

1 reference

https://api.crossref.org/works/10.1038%2FKI.1989.26

7 January 2021

Breakdown of C3 after complement activation. Identification of a new fragment C3g, using monoclonal antibodies

1 reference

https://api.crossref.org/works/10.1038%2FKI.1989.26

7 January 2021

Detection of disulphide bonds and localization of interchain linkages in the third (C3) and the fourth (C4) components of human complement

1 reference

https://api.crossref.org/works/10.1038%2FKI.1989.26

7 January 2021

Activation and fragmentation of the third (C3) and the fourth (C4) components of complement: generation and isolation of physiologically relevant fragments C3c and C4c.

1 reference

https://api.crossref.org/works/10.1038%2FKI.1989.26

7 January 2021

Abnormality of glycophorin-alpha on paroxysmal nocturnal hemoglobinuria erythrocytes

1 reference

https://api.crossref.org/works/10.1038%2FKI.1989.26

7 January 2021

Enzyme immunoassay ELISA and EMIT

1 reference

https://api.crossref.org/works/10.1038%2FKI.1989.26

7 January 2021

PEROXIDASE-LABELED ANTIBODY A NEW METHOD OF CONJUGATION

1 reference

https://api.crossref.org/works/10.1038%2FKI.1989.26

7 January 2021

Third component of human complement: purification from plasma and physicochemical characterization

1 reference

https://api.crossref.org/works/10.1038%2FKI.1989.26

7 January 2021

Human complement component C3: cDNA coding sequence and derived primary structure

1 reference

https://api.crossref.org/works/10.1038%2FKI.1989.26

7 January 2021

An autosomal dominant gene regulates the extent of 9-O-acetylation of murine erythrocyte sialic acids. A probable explanation for the variation in capacity to activate the human alternate complement pathway

1 reference

https://api.crossref.org/works/10.1038%2FKI.1989.26

7 January 2021

Surface-associated heparin inhibits zymosan-induced activation of the human alternative complement pathway by augmenting the regulatory action of the control proteins on particle-bound C3b

1 reference

https://api.crossref.org/works/10.1038%2FKI.1989.26

7 January 2021

Identifiers

DOI

10.1038/KI.1989.26

1 reference

29 September 2017

1 reference