(Q41972438)

English

Vildagliptin stimulates endothelial cell network formation and ischemia-induced revascularization via an endothelial nitric-oxide synthase-dependent mechanism.

scientific article published on 6 August 2014

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scholarly article

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12 March 2020

Vildagliptin stimulates endothelial cell network formation and ischemia-induced revascularization via an endothelial nitric-oxide synthase-dependent mechanism (English)

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12 March 2020

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revascularization

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based on heuristic

inferred from title

7

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12 March 2020

author name string

Masakazu Ishii

1

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12 March 2020

Rei Shibata

2

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12 March 2020

Kazuhisa Kondo

3

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12 March 2020

Takahiro Kambara

4

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12 March 2020

Yuuki Shimizu

5

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12 March 2020

Tohru Tanigawa

6

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12 March 2020

Masahiro Nishimura

8

1 reference

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12 March 2020

Noriyuki Ouchi

9

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12 March 2020

Toyoaki Murohara

10

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12 March 2020

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publication date

6 August 2014

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12 March 2020

Journal of Biological Chemistry

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12 March 2020

289

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12 March 2020

27235-27245

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12 March 2020

39

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12 March 2020

describes a project that uses

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11 June 2022

https://www.ebi.ac.uk/europepmc/webservices/rest/PMC4175356/fullTextXML

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Effects of combination therapy with vildagliptin and valsartan in a mouse model of type 2 diabetes.

1 reference

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Dipeptidylpeptidase inhibition is associated with improvement in blood pressure and diastolic function in insulin-resistant male Zucker obese rats

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The emerging role of dipeptidyl peptidase-4 inhibitors in cardiovascular protection: current position and perspectives

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Dipeptidyl peptidase-4 inhibition and vascular repair by mobilization of endogenous stem cells in diabetes and beyond

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The dipeptidyl peptidase-4 inhibitor sitagliptin improves vascular endothelial function in type 2 diabetes

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Diabetes and cardiovascular disease: changing the focus from glycemic control to improving long-term survival

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Dipeptidyl peptidase-4 inhibitor improves neovascularization by increasing circulating endothelial progenitor cells

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CTRP9 protein protects against myocardial injury following ischemia-reperfusion through AMP-activated protein kinase (AMPK)-dependent mechanism

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Effect of a dipeptidyl peptidase-IV inhibitor, des-fluoro-sitagliptin, on neointimal formation after balloon injury in rats

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Clinical implications of cardiovascular preventing pleiotropic effects of dipeptidyl peptidase-4 inhibitors

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Glucagon-like peptide-1 activates endothelial nitric oxide synthase in human umbilical vein endothelial cells

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Fat-derived factor omentin stimulates endothelial cell function and ischemia-induced revascularization via endothelial nitric oxide synthase-dependent mechanism

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Enhanced angiogenesis by transplantation of mesenchymal stem cell sheet created by a novel magnetic tissue engineering method

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Acute DPP-4 inhibition modulates vascular tone through GLP-1 independent pathways

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Exendin-4, a GLP-1 receptor agonist, directly induces adiponectin expression through protein kinase A pathway and prevents inflammatory adipokine expression

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Adiponectin and cardiovascular disease

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Caloric restriction stimulates revascularization in response to ischemia via adiponectin-mediated activation of endothelial nitric-oxide synthase

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Implantation of adipose-derived regenerative cells enhances ischemia-induced angiogenesis

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Adiponectin stimulates angiogenesis in response to tissue ischemia through stimulation of amp-activated protein kinase signaling

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Regulation of endothelial-type NO synthase expression in pathophysiology and in response to drugs.

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Nitric oxide synthase modulates angiogenesis in response to tissue ischemia

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A dipeptidyl peptidase-4 inhibitor, des-fluoro-sitagliptin, improves endothelial function and reduces atherosclerotic lesion formation in apolipoprotein E-deficient mice.

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Long-term dipeptidyl-peptidase 4 inhibition reduces atherosclerosis and inflammation via effects on monocyte recruitment and chemotaxis

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inferred from PubMed ID database lookup

Identifiers

10.1074/JBC.M114.557835

1 reference

Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:25100725%20AND%20SRC:MED&resulttype=core&format=json

12 March 2020

1 reference

Europe PubMed Central

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12 March 2020

1 reference

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12 March 2020

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