(Q42437284)

English

Deficiency of the mouse complement regulatory protein mCd59b results in spontaneous hemolytic anemia with platelet activation and progressive male infertility

scientific article published on February 2003

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

31 October 2017

Deficiency of the mouse complement regulatory protein mCd59b results in spontaneous hemolytic anemia with platelet activation and progressive male infertility (English)

1 reference

Europe PubMed Central

PubMed publication ID

31 October 2017

hemolytic anemia

1 reference

based on heuristic

inferred from title

author name string

Xuebin Qin

1

1 reference

Europe PubMed Central

PubMed publication ID

31 October 2017

Nicole Krumrei

2

1 reference

Europe PubMed Central

PubMed publication ID

31 October 2017

Luciano Grubissich

3

1 reference

Europe PubMed Central

PubMed publication ID

31 October 2017

Martin Dobarro

4

1 reference

Europe PubMed Central

PubMed publication ID

31 October 2017

Huseyin Aktas

5

1 reference

Europe PubMed Central

PubMed publication ID

31 October 2017

Graciela Perez

6

1 reference

Europe PubMed Central

PubMed publication ID

31 October 2017

Jose A Halperin

7

1 reference

Europe PubMed Central

PubMed publication ID

31 October 2017

language of work or name

0 references

publication date

1 February 2003

1 reference

Europe PubMed Central

PubMed publication ID

31 October 2017

1 reference

Europe PubMed Central

PubMed publication ID

31 October 2017

18

1 reference

Europe PubMed Central

PubMed publication ID

31 October 2017

2

1 reference

Europe PubMed Central

PubMed publication ID

31 October 2017

217-227

1 reference

Europe PubMed Central

PubMed publication ID

31 October 2017

Molecular basis for a link between complement and the vascular complications of diabetes

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Terminal complement proteins C5b-9 release basic fibroblast growth factor and platelet-derived growth factor from endothelial cells

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A spectrophotometric scanning technique for the rapid determination of plasma hemoglobin

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Mutational analysis of the active site and antibody epitopes of the complement-inhibitory glycoprotein, CD59

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Pathogenesis of glomerular damage in glomerulonephritis

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Release of decay-accelerating factor (DAF) from the cell membrane by phosphatidylinositol-specific phospholipase C (PIPLC). Selective modification of a complement regulatory protein

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The pig analogue of CD59 protects transgenic mouse hearts from injury by human complement

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The Relationship Between Sperm Concentration and Fertilization in vitro of Mouse Eggs

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Development of a hemolytic assay for mouse complement components in sera and the variation of their levels with age.

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Activated platelets in paroxysmal nocturnal haemoglobinuria

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Terminal complement complex C5b-9 stimulates mitogenesis in 3T3 cells

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Chronic Hemolytic Anemia with Paroxysmal Nocturnal Hemoglobinuria

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Targeted deletion of the CD59 gene causes spontaneous intravascular hemolysis and hemoglobinuria

1 reference

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Mouse complement regulatory protein Crry/p65 uses the specific mechanisms of both human decay-accelerating factor and membrane cofactor protein

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A family showing inheritance of the Inab phenotype

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Role of CD59 in experimental glomerulonephritis in rats

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Cloning and characterization of cDNAs encoding the complete sequence of decay-accelerating factor of human complement

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Crry, but not CD59 and DAF, is indispensable for murine erythrocyte protection in vivo from spontaneous complement attack

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The effects of functional suppression of a membrane-bound complement regulatory protein, CD59, in the synovial tissue in rats

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Complement regulatory molecules: application to therapy and transplantation.

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Membrane signaling by complement C5b-9, the membrane attack complex

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Molecular basis of paroxysmal nocturnal hemoglobinuria

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Identification and functional characterization of a new gene encoding the mouse terminal complement inhibitor CD59

1 reference

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7 January 2021

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Genomic structure, functional comparison, and tissue distribution of mouse Cd59a and Cd59b

1 reference

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7 January 2021

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Physiologic relevance of the membrane attack complex inhibitory protein CD59 in human seminal plasma: CD59 is present on extracellular organelles (prostasomes), binds cell membranes, and inhibits complement-mediated lysis

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The complement system in atherosclerosis

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Complement (C5b-9) induces glomerular epithelial cell DNA synthesis but not proliferation in vitro

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Complement-mediated neurotoxicity is regulated by homologous restriction.

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Role of decay-accelerating factor in regulating complement activation on the erythrocyte surface as revealed by gene targeting

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Deficiency of the GPI anchor caused by a somatic mutation of the PIG-A gene in paroxysmal nocturnal hemoglobinuria

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C5b-8 and C5b-9 modulate the collagen release of human glomerular epithelial cells

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Complement-induced release of monocyte chemotactic protein-1 from human smooth muscle cells. A possible initiating event in atherosclerotic lesion formation

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Deficient surface expression of glycosylphosphatidylinositol-anchored proteins in B cell lines established from patients with paroxysmal nocturnal hemoglobinuria

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Estimation of classical pathway of mouse complement activity by use of sensitized rabbit erythrocytes

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A critical role for murine complement regulator crry in fetomaternal tolerance

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Inherited complete deficiency of 20-kilodalton homologous restriction factor (CD59) as a cause of paroxysmal nocturnal hemoglobinuria

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Deficiency of complement defense protein CD59 may contribute to neurodegeneration in Alzheimer's disease

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Identifiers

10.1016/S1074-7613(03)00022-0

1 reference

Europe PubMed Central

PubMed publication ID

31 October 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

31 October 2017

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