(Q42458805)

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Computational identification of potential transcriptional regulators of TGF-ß1 in human atherosclerotic arteries.

scientific article published on 10 May 2014

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18 March 2020

Computational identification of potential transcriptional regulators of TGF-ß1 in human atherosclerotic arteries (English)

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18 March 2020

atherosclerosis

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based on heuristic

inferred from title

Giampiero Bricca

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18 March 2020

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Nedra Dhaouadi

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18 March 2020

Jacques-Yuan Li

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18 March 2020

Patrick Feugier

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18 March 2020

Marie-Paule Gustin

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Houcine Dab

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18 March 2020

Kamel Kacem

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18 March 2020

Catherine Cerutti

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18 March 2020

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10 May 2014

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18 March 2020

357-370

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18 March 2020

Cluster analysis and display of genome-wide expression patterns

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Transcriptional activation of transforming growth factor beta1 and its receptors by the Kruppel-like factor Zf9/core promoter-binding protein and Sp1. Potential mechanisms for autocrine fibrogenesis in response to injury.

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Angiotensin II induces human TGF-beta 1 promoter activation: similarity to hyperglycaemia

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Requirement of Ras/MAPK pathway activation by transforming growth factor beta for transforming growth factor beta 1 production in a Smad-dependent pathway

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Transactivation via RAR/RXR-Sp1 interaction: characterization of binding between Sp1 and GC box motif

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Upstream stimulatory factor (USF) proteins induce human TGF-beta1 gene activation via the glucose-response element-1013/-1002 in mesangial cells: up-regulation of USF activity by the hexosamine biosynthetic pathway

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NF-kappaB and activator protein 1 response elements and the role of histone modifications in IL-1beta-induced TGF-beta1 gene transcription

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Stretch augments TGF-beta1 expression through RhoA/ROCK1/2, PTK, and PI3K in airway smooth muscle cells

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GLI2 regulates TGF-β1 in human CD4+ T cells: implications in cancer and HIV pathogenesis

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Functional meta-analysis of double connectivity in gene coexpression networks in mammals

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Natural history and histological classification of atherosclerotic lesions: an update

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A definition of the intima of human arteries and of its atherosclerosis-prone regions. A report from the Committee on Vascular Lesions of the Council on Arteriosclerosis, American Heart Association

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Loss of Akt1 leads to severe atherosclerosis and occlusive coronary artery disease

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Absence of Akt1 reduces vascular smooth muscle cell migration and survival and induces features of plaque vulnerability and cardiac dysfunction during atherosclerosis.

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Akt interacts directly with Smad3 to regulate the sensitivity to TGF-beta induced apoptosis

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PKB/Akt modulates TGF-beta signalling through a direct interaction with Smad3.

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Novel roles of Akt and mTOR in suppressing TGF-beta/ALK5-mediated Smad3 activation

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BAT3 interacts with transforming growth factor-beta (TGF-beta) receptors and enhances TGF-beta1-induced type I collagen expression in mesangial cells

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Transcriptional activation of endoglin and transforming growth factor-beta signaling components by cooperative interaction between Sp1 and KLF6: their potential role in the response to vascular injury

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