(Q42481896)

1 November 2017

Smad3 signalling plays an important role in keloid pathogenesis via epithelial-mesenchymal interactions (English)

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1 November 2017

Phan TT

1

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1 November 2017

Lim IJ

2

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1 November 2017

Aalami O

3

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1 November 2017

Lorget F

4

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1 November 2017

Khoo A

5

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1 November 2017

Tan EK

6

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1 November 2017

Mukhopadhyay A

7

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1 November 2017

Longaker MT

8

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1 November 2017

1 October 2005

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1 November 2017

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1 November 2017

207

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1 November 2017

232-242

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1 November 2017

2

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Fibroblasts cocultured with keloid keratinocytes: normal fibroblasts secrete collagen in a keloidlike manner

1 November 2017

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Role of IGF system of mitogens in the induction of fibroblast proliferation by keloid-derived keratinocytes in vitro

21 January 2018

1 reference

Increased glomerular and tubular expression of transforming growth factor-beta1, its type II receptor, and activation of the Smad signaling pathway in the db/db mouse

21 January 2018

1 reference

Smad3 deficiency attenuates bleomycin-induced pulmonary fibrosis in mice

21 January 2018

1 reference

Smad3 null mice develop airspace enlargement and are resistant to TGF-beta-mediated pulmonary fibrosis

21 January 2018

1 reference

Smad3 signaling is required for epithelial-mesenchymal transition of lens epithelium after injury

21 January 2018

1 reference

Smad3 and PKCdelta mediate TGF-beta1-induced collagen I expression in human mesangial cells

21 January 2018

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Phosphatidylinositol 3-kinase is involved in alpha2(I) collagen gene expression in normal and scleroderma fibroblasts

21 January 2018

1 reference

Targeted disruption of TGF-beta/Smad3 signaling modulates skin fibrosis in a mouse model of scleroderma

21 January 2018

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Mice lacking Smad3 are protected against cutaneous injury induced by ionizing radiation

21 January 2018

1 reference

Interference with transforming growth factor-beta/ Smad3 signaling results in accelerated healing of wounds in previously irradiated skin

21 January 2018

1 reference

Deficient Smad7 expression: a putative molecular defect in scleroderma

21 January 2018

1 reference

9 February 2024

https://opencitations.net/index/api/v1/citations/10.1073/PNAS.062010399

Identifiers

DOI

10.1002/PATH.1826

1 reference

1 November 2017

1 reference