(Q42494967)

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Dysfunction of M-channel enhances propagation of neuronal excitability in rat hippocampus monitored by multielectrode dish and microdialysis systems.

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:11044585%20AND%20SRC:MED&resulttype=core&format=json

1 November 2019

Dysfunction of M-channel enhances propagation of neuronal excitability in rat hippocampus monitored by multielectrode dish and microdialysis systems (English)

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Europe PubMed Central

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:11044585%20AND%20SRC:MED&resulttype=core&format=json

1 November 2019

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1 November 2019

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G Zhu

1

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1 November 2019

T Murakami

3

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1 November 2019

A Kamata

4

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1 November 2019

Y Kawata

5

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1 November 2019

K Wada

6

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1 November 2019

S Kaneko

7

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1 November 2019

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1 November 2000

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1 November 2019

Neuroscience Letters

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1 November 2019

294

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1 November 2019

1

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1 November 2019

53-57

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1 November 2019

A potassium channel mutation in neonatal human epilepsy

1 reference

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Molecular cloning and functional expression of KCNQ5, a potassium channel subunit that may contribute to neuronal M-current diversity

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Excitatory GABA responses in embryonic and neonatal cortical slices demonstrated by gramicidin perforated-patch recordings and calcium imaging.

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Moderate loss of function of cyclic-AMP-modulated KCNQ2/KCNQ3 K+ channels causes epilepsy

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A novel potassium channel gene, KCNQ2, is mutated in an inherited epilepsy of newborns

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KCNQ2 and KCNQ3 potassium channel subunits: molecular correlates of the M-channel

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Disruption of the epilepsy KCNQ2 gene results in neural hyperexcitability

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inferred from DOI database lookup

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