(Q42496768)

1 November 2017

title

Defective electron transfer in complexes I and IV in patients with aceruloplasminemia (English)

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1 November 2017

Kohno S

1

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1 November 2017

Miyajima H

2

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1 November 2017

Takahashi Y

3

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1 November 2017

Suzuki H

4

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1 November 2017

Hishida A

5

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1 November 2017

publication date

1 December 2000

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Journal of the Neurological Sciences

1 November 2017

published in

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1 November 2017

volume

182

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1 November 2017

issue

1

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1 November 2017

page(s)

57-60

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A mutation in the ceruloplasmin gene is associated with systemic hemosiderosis in humans

1 November 2017

cites work

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Aceruloplasminemia: molecular characterization of this disorder of iron metabolism

7 January 2021

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Hereditary ceruloplasmin deficiency with hemosiderosis: a clinicopathological study of a Japanese family

7 January 2021

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Radical reactions from missing ceruloplasmin: the importance of a ferroxidase as an endogenous antioxidant

7 January 2021

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Increased plasma lipid peroxidation in patients with aceruloplasminemia

7 January 2021

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7 January 2021

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Familial apoceruloplasmin deficiency associated with blepharospasm and retinal degeneration

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Age-dependent impairment of mitochondrial function in primate brain.

7 January 2021

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On the site of action of the inhibition of the mitochondrial respiratory chain by lipoxygenase

7 January 2021

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Alteration of inner-membrane components and damage to electron-transfer activities of bovine heart submitochondrial particles induced by NADPH-dependent lipid peroxidation

7 January 2021

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Normal oxidative damage to mitochondrial and nuclear DNA is extensive

7 January 2021

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Evidence for mitochondrial DNA damage by lipid peroxidation

7 January 2021

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Sequential damage in mitochondrial complexes by peroxidative stress

7 January 2021

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Role of mitochondria in oxidative stress and ageing

7 January 2021

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Mitochondria in neurodegenerative apoptosis: an opportunity for therapy?

7 January 2021

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Bioenergetic and oxidative stress in neurodegenerative diseases

7 January 2021

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Mitochondrial involvement in Parkinson's disease, Huntington's disease, hereditary spastic paraplegia and Friedreich's ataxia

7 January 2021

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https://api.crossref.org/works/10.1016%2FS0022-510X%2800%2900452-4

10.1016/S0022-510X(00)00452-4

7 January 2021

Identifiers

DOI

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1 November 2017

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