(Q42501032)

English

9-cis-Retinoic acid represses transcription of the gonadotropin-releasing hormone (GnRH) gene via proximal promoter region that is distinct from all-trans-retinoic acid response element

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scholarly article

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Europe PubMed Central

PubMed publication ID

1 November 2017

9-cis-Retinoic acid represses transcription of the gonadotropin-releasing hormone (GnRH) gene via proximal promoter region that is distinct from all-trans-retinoic acid response element (English)

1 reference

Europe PubMed Central

PubMed publication ID

1 November 2017

1 reference

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author name string

S Cho

1

1 reference

Europe PubMed Central

PubMed publication ID

1 November 2017

J Chung

2

1 reference

Europe PubMed Central

PubMed publication ID

1 November 2017

J Han

3

1 reference

Europe PubMed Central

PubMed publication ID

1 November 2017

B Ju Lee

4

1 reference

Europe PubMed Central

PubMed publication ID

1 November 2017

D Han Kim

5

1 reference

Europe PubMed Central

PubMed publication ID

1 November 2017

K Rhee

6

1 reference

Europe PubMed Central

PubMed publication ID

1 November 2017

K Kim

7

1 reference

Europe PubMed Central

PubMed publication ID

1 November 2017

publication date

1 March 2001

1 reference

Europe PubMed Central

PubMed publication ID

1 November 2017

Molecular Brain Research

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Europe PubMed Central

PubMed publication ID

1 November 2017

87

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Europe PubMed Central

PubMed publication ID

1 November 2017

2

1 reference

Europe PubMed Central

PubMed publication ID

1 November 2017

214-222

1 reference

Europe PubMed Central

PubMed publication ID

1 November 2017

Retinoic acid receptors and retinoid X receptors: interactions with endogenous retinoic acids

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9-cis-retinoic acid is a natural antagonist for the retinoic acid receptor response pathway

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The glucocorticoid receptor is tethered to DNA-bound Oct-1 at the mouse gonadotropin-releasing hormone distal negative glucocorticoid response element

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Retinoic acid regulates gonadotropin-releasing hormone (GnRH) release and gene expression in the rat hypothalamic fragments and GT1-1 neuronal cells in vitro

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Evidence for autocrine inhibition of gonadotropin-releasing hormone (GnRH) gene transcription by GnRH in hypothalamic GT1-1 neuronal cells

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The opposing effects of retinoic acid and phorbol esters converge to a common response element in the promoter of the rat cholesterol 7 alpha-hydroxylase gene (CYP7A).

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Isolation and functional characterization of the human gene encoding the myeloid zinc finger protein MZF-1.

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Direct binding of progesterone receptor to nonconsensus DNA sequences represses rat GnRH.

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Formation of retinoid X receptor homodimers leads to repression of T3 response: hormonal cross talk by ligand-induced squelching

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Differential effects of retinoic acid isomers on the expression of nuclear receptor co-regulators in neuroblastoma

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A deletion truncating the gonadotropin-releasing hormone gene is responsible for hypogonadism in the hpg mouse.

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Retinoid receptors in transcriptional regulation

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Characterization of the DNA-binding properties of the myeloid zinc finger protein MZF1: two independent DNA-binding domains recognize two DNA consensus sequences with a common G-rich core

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The GnRH promoter: target of transcription factors, hormones, and signaling pathways

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Differential effects of 9-cis and all-trans retinoic acid on the induction of retinoic acid receptor-beta and cellular retinoic acid-binding protein II in human neuroblastoma cells

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Retinoids in embryonal development

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Retinoid X receptors

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Activation of heat shock gene transcription by heat shock factor 1 involves oligomerization, acquisition of DNA-binding activity, and nuclear localization and can occur in the absence of stress

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The discovery of 9-cis retinoic acid: a hormone that binds the retinoid-X receptor

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Identifiers

10.1016/S0169-328X(01)00020-1

1 reference

Europe PubMed Central

PubMed publication ID

1 November 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

1 November 2017

ResearchGate publication ID

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