(Q42503629)

1 November 2017

title

Modulation of SR Ca2+ release by the triadin-to-calsequestrin ratio in ventricular myocytes (English)

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1 November 2017

Dana Kučerová

1

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1 November 2017

Hideo A Baba

2

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1 November 2017

Peter Bokník

3

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1 November 2017

Larissa Fabritz

4

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1 November 2017

Alexander Heinick

5

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1 November 2017

Marek Mát'uš

6

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1 November 2017

Frank U Müller

7

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1 November 2017

Joachim Neumann

8

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1 November 2017

Wilhelm Schmitz

9

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1 November 2017

Uwe Kirchhefer

10

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1 November 2017

publication date

16 March 2012

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American Journal of Physiology Heart and Circulatory Physiology

1 November 2017

published in

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1 November 2017

volume

302

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1 November 2017

issue

10

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1 November 2017

page(s)

H2008-17

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Macromolecular complexes regulating cardiac ryanodine receptor function.

1 November 2017

cites work

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00457.2011

Ablation of triadin causes loss of cardiac Ca2+ release units, impaired excitation-contraction coupling, and cardiac arrhythmias

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00457.2011

L-type Ca2+ current downregulation in chronic human atrial fibrillation is associated with increased activity of protein phosphatases

21 January 2018

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https://api.crossref.org/works/10.1152%2FAJPHEART.00457.2011

Architecture and regulation of the Ca2+ delivery system in muscle cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00457.2011

On the role of junctin in cardiac Ca2+ handling, contractility, and heart failure

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00457.2011

The role of calsequestrin, triadin, and junctin in conferring cardiac ryanodine receptor responsiveness to luminal calcium

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00457.2011

Modulation of ryanodine receptor by luminal calcium and accessory proteins in health and cardiac disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00457.2011

Calcium handling proteins in the failing human heart

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00457.2011

Regulation of Ca2+ signaling in transgenic mouse cardiac myocytes overexpressing calsequestrin

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00457.2011

Transgenic triadin 1 overexpression alters SR Ca2+ handling and leads to a blunted contractile response to beta-adrenergic agonists

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00457.2011

Cardiac hypertrophy and impaired relaxation in transgenic mice overexpressing triadin 1

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00457.2011

Impaired relaxation in transgenic mice overexpressing junctin.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00457.2011

The human CASQ2 mutation K206N is associated with hyperglycosylation and altered cellular calcium handling

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00457.2011

Localization and characterization of the calsequestrin-binding domain of triadin 1. Evidence for a charged beta-strand in mediating the protein-protein interaction

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00457.2011

Identification of triadin 1 as the predominant triadin isoform expressed in mammalian myocardium

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00457.2011

Isoproterenol-induced heart failure in the rat is associated with nitric oxide-dependent functional alterations of cardiac function

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00457.2011

Abnormalities of calcium metabolism and myocardial contractility depression in the failing heart

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00457.2011

Critical role of transcription factor cyclic AMP response element modulator in beta1-adrenoceptor-mediated cardiac dysfunction.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00457.2011

Polymerization of calsequestrin. Implications for Ca2+ regulation

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00457.2011

Improved resolution of myofibrillar proteins with sodium dodecyl sulfate-polyacrylamide gel electrophoresis

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00457.2011

Cardiac and skeletal muscle disorders caused by mutations in the intracellular Ca2+ release channels.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00457.2011

Cardiac-specific overexpression of mouse cardiac calsequestrin is associated with depressed cardiovascular function and hypertrophy in transgenic mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00457.2011

Protein protein interactions between triadin and calsequestrin are involved in modulation of sarcoplasmic reticulum calcium release in cardiac myocytes

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00457.2011

Complex formation between junctin, triadin, calsequestrin, and the ryanodine receptor. Proteins of the cardiac junctional sarcoplasmic reticulum membrane

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00457.2011

Stress and high heart rate provoke ventricular tachycardia in mice expressing triadin

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00457.2011

10.1152/AJPHEART.00457.2011

21 January 2018

Identifiers

DOI

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1 November 2017

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