(Q42528299)

English

Specific up-regulation of CRH or AVP secretion by acetylcholine or lipopolysaccharide in inflammatory susceptible Lewis rat fetal hypothalamic cells

scientific article published on October 2002

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

1 November 2017

Specific up-regulation of CRH or AVP secretion by acetylcholine or lipopolysaccharide in inflammatory susceptible Lewis rat fetal hypothalamic cells (English)

1 reference

Europe PubMed Central

PubMed publication ID

1 November 2017

lipopolysaccharide

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author name string

Rongtai Wei

1

1 reference

Europe PubMed Central

PubMed publication ID

1 November 2017

Terry M Phillips

2

1 reference

Europe PubMed Central

PubMed publication ID

1 November 2017

Esther M Sternberg

3

1 reference

Europe PubMed Central

PubMed publication ID

1 November 2017

publication date

1 October 2002

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Europe PubMed Central

PubMed publication ID

1 November 2017

Journal of Neuroimmunology

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Europe PubMed Central

PubMed publication ID

1 November 2017

131

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Europe PubMed Central

PubMed publication ID

1 November 2017

1-2

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Europe PubMed Central

PubMed publication ID

1 November 2017

31-40

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Europe PubMed Central

PubMed publication ID

1 November 2017

Optimized survival of hippocampal neurons in B27-supplemented Neurobasal, a new serum-free medium combination

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Neurotransmitter-induced hypothalamic-pituitary-adrenal axis responsiveness is defective in inflammatory disease-susceptible Lewis rats: in vivo and in vitro studies suggesting globally defective hypothalamic secretion of corticotropin-releasing hor

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Comparison of peptide release from fetal rat hypothalamic neurones cultured in defined media and serum-containing media.

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Repeated Stress-Induced Activation of Corticotropin-Releasing Factor Neurons Enhances Vasopressin Stores and Colocalization with Corticotropin-Releasing Factor in the Median Eminence of Rats

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Second messengers involved in the regulation of corticotropin-releasing hormone mRNA and peptide in cultured rat fetal hypothalamic primary cultures

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Corticotropin releasing activity of the new CRF is potentiated several times by vasopressin

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Interleukin-1 beta induces corticotropin-releasing factor-41 release from cultured hypothalamic cells through protein kinase C and cAMP-dependent protein kinase pathways

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Conditioned medium from activated microglia promotes cholinergic differentiation in the basal forebrain in vitro

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Temporal profiles of interleukin-1beta, interleukin-6, and tumor necrosis factor-alpha in the plasma and hypothalamic paraventricular nucleus after intravenous or intraperitoneal administration of lipopolysaccharide in the rat: estimation by push-pu

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Systemic endotoxin increases steady-state gene expression of hypothalamic nitric oxide synthase: comparison with corticotropin-releasing factor and vasopressin gene transcripts

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Interleukins-1 and -6 Stimulate the Release of Corticotropin-Releasing Hormone-41 from Rat Hypothalamusin Vitrovia the Eicosanoid Cyclooxygenase Pathway*

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Stimulation by interleukin-1 (IL-1) of the release of rat corticotropin-releasing factor (CRF), which is independent of the cholinergic mechanism, from superfused rat hypothalamo-neurohypophysial complexes

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IL-2 induces vasopressin release from the hypothalamus and the amygdala: role of nitric oxide-mediated signaling

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Reduced corticotropin-releasing factor and enhanced vasopressin gene expression in brains of mice with autoimmunity-induced behavioral dysfunction.

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Cytokines Stimulate the CRH but Not the Vasopressin Neuronal System: Evidence for a Median Eminence Site of Interleukin-6 Action

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Inflammatory mediator-induced hypothalamic-pituitary-adrenal axis activation is defective in streptococcal cell wall arthritis-susceptible Lewis rats

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A central nervous system defect in biosynthesis of corticotropin-releasing hormone is associated with susceptibility to streptococcal cell wall-induced arthritis in Lewis rats

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Identifiers

10.1016/S0165-5728(02)00251-5

1 reference

Europe PubMed Central

PubMed publication ID

1 November 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

1 November 2017

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