(Q42689668)

English

Several cardiomyopathy causing mutations on tropomyosin either destabilize the active state of actomyosin or alter the binding properties of tropomyosin

scientific article published on 3 February 2011

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scholarly article

1 reference

Europe PubMed Central

PMC publication ID

4 November 2017

Several cardiomyopathy causing mutations on tropomyosin either destabilize the active state of actomyosin or alter the binding properties of tropomyosin (English)

1 reference

Europe PubMed Central

PMC publication ID

4 November 2017

1 reference

based on heuristic

inferred from title

0 references

author name string

Mohit C Mathur

1

1 reference

Europe PubMed Central

PMC publication ID

4 November 2017

P Bryant Chase

2

1 reference

Europe PubMed Central

PMC publication ID

4 November 2017

Joseph M Chalovich

3

1 reference

Europe PubMed Central

PMC publication ID

4 November 2017

language of work or name

0 references

publication date

3 February 2011

1 reference

Europe PubMed Central

PMC publication ID

4 November 2017

Biochemical and Biophysical Research Communications

1 reference

Europe PubMed Central

PMC publication ID

4 November 2017

406

1 reference

Europe PubMed Central

PMC publication ID

4 November 2017

1

1 reference

Europe PubMed Central

PMC publication ID

4 November 2017

74-78

1 reference

Europe PubMed Central

PMC publication ID

4 November 2017

Some cardiomyopathy-causing troponin I mutations stabilize a functional intermediate actin state.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3057449

27 May 2018

Negative charges at protein kinase C sites of troponin I stabilize the inactive state of actin

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3057449

27 May 2018

A comparison of muscle thin filament models obtained from electron microscopy reconstructions and low-angle X-ray fibre diagrams from non-overlap muscle

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3057449

27 May 2018

An atomic model of the thin filament in the relaxed and Ca2+-activated states.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3057449

27 May 2018

The genetic basis for cardiac remodeling

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3057449

27 May 2018

Single particle analysis of relaxed and activated muscle thin filaments.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3057449

27 May 2018

The Delta 14 mutation of human cardiac troponin T enhances ATPase activity and alters the cooperative binding of S1-ADP to regulated actin.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3057449

27 May 2018

Effects of two familial hypertrophic cardiomyopathy mutations in alpha-tropomyosin, Asp175Asn and Glu180Gly, on the thermal unfolding of actin-bound tropomyosin

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3057449

27 May 2018

American College of Cardiology/European Society of Cardiology clinical expert consensus document on hypertrophic cardiomyopathy. A report of the American College of Cardiology Foundation Task Force on Clinical Expert Consensus Documents and the Euro

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3057449

27 May 2018

Regulation of striated muscle contraction: a discussion

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3057449

27 May 2018

Mechanism of regulation of phosphate dissociation from actomyosin-ADP-Pi by thin filament proteins

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3057449

27 May 2018

Hypertrophic cardiomyopathy: a systematic review

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3057449

27 May 2018

The Genetic Basis for Cardiomyopathy

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3057449

27 May 2018

Hypertrophic cardiomyopathy caused by a novel alpha-tropomyosin mutation (V95A) is associated with mild cardiac phenotype, abnormal calcium binding to troponin, abnormal myosin cycling, and poor prognosis

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3057449

27 May 2018

Regulation of the interaction between actin and myosin subfragment 1: evidence for three states of the thin filament

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3057449

27 May 2018

The mechanism of regulation of actomyosin subfragment 1 ATPase

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3057449

27 May 2018

Actin mediated regulation of muscle contraction

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3057449

27 May 2018

Separation of subfragment-1 isoenzymes from rabbit skeletal muscle myosin

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3057449

27 May 2018

Ca2+ sensitivity of regulated cardiac thin filament sliding does not depend on myosin isoform.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3057449

16 August 2018

Cardiac dysfunction in hypertrophic cardiomyopathy mutant tropomyosin mice is transgene-dependent, hypertrophy-independent, and improved by beta-blockade

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3057449

16 August 2018

The genetic basis for cardiomyopathy: from mutation identification to mechanistic paradigms

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3057449

16 August 2018

Effects of two familial hypertrophic cardiomyopathy-causing mutations on alpha-tropomyosin structure and function.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3057449

16 August 2018

Cooperative interactions between adjacent troponin-tropomyosin complexes may be transmitted through the actin filament

1 reference

https://pubmed.ncbi.nlm.nih.gov/21295541

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

The active state of the thin filament is destabilized by an internal deletion in tropomyosin

1 reference

https://pubmed.ncbi.nlm.nih.gov/21295541

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Mouse model of a familial hypertrophic cardiomyopathy mutation in alpha-tropomyosin manifests cardiac dysfunction

1 reference

https://pubmed.ncbi.nlm.nih.gov/21295541

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

A model for the myosin molecule

1 reference

https://pubmed.ncbi.nlm.nih.gov/21295541

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1016/J.BBRC.2011.01.112

1 reference

Europe PubMed Central

PMC publication ID

4 November 2017

PMC publication ID

1 reference

Europe PubMed Central

PMC publication ID

4 November 2017

PubMed publication ID

1 reference

Europe PubMed Central

PMC publication ID

4 November 2017

ResearchGate publication ID

0 references

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