(Q42798672)

7 November 2017

The transformation suppressor protein Pdcd4 shuttles between nucleus and cytoplasm and binds RNA. (English)

1 reference

7 November 2017

Maret Böhm

1

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7 November 2017

Kirsty Sawicka

2

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7 November 2017

Jan Peter Siebrasse

3

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7 November 2017

Anne Brehmer-Fastnacht

4

1 reference

7 November 2017

Reiner Peters

5

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7 November 2017

Karl-Heinz Klempnauer

6

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7 November 2017

language of work or name

English

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publication date

1 July 2003

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7 November 2017

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7 November 2017

22

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7 November 2017

4905-4910

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7 November 2017

31

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https://scigraph.springernature.com/pub.10.1038/sj.onc.1206710

7 November 2017

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cites work

Differentially expressed protein Pdcd4 inhibits tumor promoter-induced neoplastic transformation

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206710

Evidence for specific nucleocytoplasmic transport pathways used by leucine-rich nuclear export signals

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206710

DUG is a novel homologue of translation initiation factor 4G that binds eIF4A

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206710

Up-regulation of PDCD4 in senescent human diploid fibroblasts

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206710

Integration of splicing, transport and translation to achieve mRNA quality control by the nonsense-mediated decay pathway

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206710

Targeted disruption of c-myb in the chicken pre B-cell line DT40.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206710

7 January 2021

CRM1 is an export receptor for leucine-rich nuclear export signals

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206710

7 January 2021

CRM1 is responsible for intracellular transport mediated by the nuclear export signal

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206710

7 January 2021

A comparison of the activity, sequence specificity, and CRM1-dependence of different nuclear export signals

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206710

7 January 2021

A perfect message: RNA surveillance and nonsense-mediated decay

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206710

7 January 2021

Transformation by v-Myb

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206710

7 January 2021

Quality control of mRNA function

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206710

7 January 2021

Continuous tissue culture cell lines derived from chemically induced tumors of Japanese quail

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206710

7 January 2021

The myb gene family in cell growth, differentiation and apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206710

7 January 2021

The chicken Pdcd4 gene is regulated by v-Myb

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206710

7 January 2021

Identification of the myb-inducible promoter of the chicken Pdcd4 gene

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206710

7 January 2021

Isolation of a novel mouse gene MA-3 that is induced upon programmed cell death

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206710

7 January 2021

RNA recognition by arginine-rich peptide motifs

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206710

7 January 2021

Identification of a signal for rapid export of proteins from the nucleus

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206710

7 January 2021

Reassessing the role of C-MYB in tumorigenesis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206710

7 January 2021

A novel transformation suppressor, Pdcd4, inhibits AP-1 transactivation but not NF-kappaB or ODC transactivation

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206710

7 January 2021

Identifiers

DOI

10.1038/SJ.ONC.1206710

1 reference

Dimensions Publication ID

7 November 2017

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1 reference