(Q42829924)

8 November 2017

title

Steroid receptor fusion proteins for conditional activation of Raf-MEK-ERK signaling pathway (English)

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8 November 2017

M McMahon

1

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8 November 2017

language of work or name

English

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publication date

1 January 2001

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8 November 2017

published in

Methods in Enzymology

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8 November 2017

volume

332

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8 November 2017

page(s)

401-417

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Chimaeras of Myc oncoprotein and steroid receptors cause hormone-dependent transformation of cells

8 November 2017

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Conditional transformation of cells and rapid activation of the mitogen-activated protein kinase cascade by an estradiol-dependent human raf-1 protein kinase

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7 January 2021

Activated MEK stimulates expression of AP-1 components independently of phosphatidylinositol 3-kinase (PI3-kinase) but requires a PI3-kinase signal To stimulate DNA synthesis

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Hormone-conditional transformation by fusion proteins of c-Abl and its transforming variants

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CDC25A phosphatase is a target of E2F and is required for efficient E2F-induced S phase

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A modified oestrogen receptor ligand-binding domain as an improved switch for the regulation of heterologous proteins

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Rapid phosphorylation of Ets-2 accompanies mitogen-activated protein kinase activation and the induction of heparin-binding epidermal growth factor gene expression by oncogenic Raf-1.

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Rapid induction of heparin-binding epidermal growth factor/diphtheria toxin receptor expression by Raf and Ras oncogenes.

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Conditionally oncogenic forms of the A-Raf and B-Raf protein kinases display different biological and biochemical properties in NIH 3T3 cells

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Inhibition of platelet-derived growth factor- and epidermal growth factor-mediated mitogenesis and signaling in 3T3 cells expressing delta Raf-1:ER, an estradiol-regulated form of Raf-1.

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Critical tyrosine residues regulate the enzymatic and biological activity of Raf-1 kinase

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Activation of the Raf-1 kinase cascade by coumermycin-induced dimerization.

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Oligomerization activates c-Raf-1 through a Ras-dependent mechanism

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Activation of phosphatidylinositol 3-kinase is sufficient for cell cycle entry and promotes cellular changes characteristic of oncogenic transformation

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7 January 2021

Construction and characterization of a conditionally active version of the serine/threonine kinase Akt.

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7 January 2021

10.1016/S0076-6879(01)32218-8

Identifiers

DOI

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8 November 2017

1 reference