(Q42917683)

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Comparative evaluation of hypoxic–ischemic brain injury by flow cytometric analysis of mitochondrial membrane potential with JC-1 in neonatal rats

scientific article published on September 15, 2010

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11 November 2017

Comparative evaluation of hypoxic-ischemic brain injury by flow cytometric analysis of mitochondrial membrane potential with JC-1 in neonatal rats (English)

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PubMed publication ID

11 November 2017

Comparative evaluation of hypoxic–ischemic brain injury by flow cytometric analysis of mitochondrial membrane potential with JC-1 in neonatal rats (English)

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10.1016/J.JNEUMETH.2010.08.020

https://api.crossref.org/works/10.1016/J.JNEUMETH.2010.08.020

6 July 2024

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6 July 2024

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6 July 2024

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6 July 2024

author name string

Dong Kyung Sung

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11 November 2017

Yun Sil Chang

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11 November 2017

Sam Kang

3

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11 November 2017

Hwa Young Song

4

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11 November 2017

Won Soon Park

5

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11 November 2017

Bong Hee Lee

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11 November 2017

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15 September 2010

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11 November 2017

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https://api.elsevier.com/content/article/PII:S0165027010004802?httpAccept=text/xml

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10.1016/J.JNEUMETH.2010.08.020

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Journal of Neuroscience Methods

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11 November 2017

193

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11 November 2017

2

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11 November 2017

232-238

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11 November 2017

Glutamate-induced neuronal death: A succession of necrosis or apoptosis depending on mitochondrial function

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Mitochondrial cytochrome c release in apoptosis occurs upstream of DEVD-specific caspase activation and independently of mitochondrial transmembrane depolarization.

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Automated measurement of infarct size with scanned images of triphenyltetrazolium chloride-stained rat brains

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Granulocyte stimulating factor attenuates hypoxic-ischemic brain injury by inhibiting apoptosis in neonatal rats.

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Mitochondrial control of apoptosis

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The mitochondrial permeability transition: from biochemical curiosity to pathophysiological mechanism

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Characterization of cells with different mitochondrial membrane potential during apoptosis

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Mitochondria and neuronal survival

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Therapeutic window for cycloheximide treatment after hypoxic-ischemic brain injury in neonatal rats

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Mitochondria and programmed cell death: back to the future.

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Term infants with hypoxic-ischemic encephalopathy: outcome at 3.5 years

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JC-1, but not DiOC6(3) or rhodamine 123, is a reliable fluorescent probe to assess delta psi changes in intact cells: implications for studies on mitochondrial functionality during apoptosis

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Ultrastructural changes of neuronal mitochondria after transient and permanent cerebral ischemia

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Identification of necrotic cell death by the TUNEL assay in the hypoxic-ischemic neonatal rat brain

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10.1016/J.JNEUMETH.2010.08.020

1 reference

Europe PubMed Central

PubMed publication ID

11 November 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

11 November 2017

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