(Q42943347)

English

Postconditioning effect of granulocyte colony-stimulating factor is mediated through activation of risk pathway and opening of the mitochondrial KATP channels

scientific article published on August 6, 2010

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Europe PubMed Central

PubMed publication ID

11 November 2017

Postconditioning effect of granulocyte colony-stimulating factor is mediated through activation of risk pathway and opening of the mitochondrial KATP channels (English)

1 reference

Europe PubMed Central

PubMed publication ID

11 November 2017

author name string

Shohei Sumi

1

1 reference

Europe PubMed Central

PubMed publication ID

11 November 2017

Hiroyuki Kobayashi

2

1 reference

Europe PubMed Central

PubMed publication ID

11 November 2017

Shinji Yasuda

3

1 reference

Europe PubMed Central

PubMed publication ID

11 November 2017

Masamitsu Iwasa

4

1 reference

Europe PubMed Central

PubMed publication ID

11 November 2017

Takahiko Yamaki

5

1 reference

Europe PubMed Central

PubMed publication ID

11 November 2017

Yoshihisa Yamada

6

1 reference

Europe PubMed Central

PubMed publication ID

11 November 2017

Hiroaki Ushikoshi

7

1 reference

Europe PubMed Central

PubMed publication ID

11 November 2017

Arihiro Hattori

8

1 reference

Europe PubMed Central

PubMed publication ID

11 November 2017

Takuma Aoyama

9

1 reference

Europe PubMed Central

PubMed publication ID

11 November 2017

Kazuhiko Nishigaki

10

1 reference

Europe PubMed Central

PubMed publication ID

11 November 2017

Genzou Takemura

11

1 reference

Europe PubMed Central

PubMed publication ID

11 November 2017

Shinya Minatoguchi

12

1 reference

Europe PubMed Central

PubMed publication ID

11 November 2017

language of work or name

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publication date

6 August 2010

1 reference

Europe PubMed Central

PubMed publication ID

11 November 2017

full work available at URL

http://intl-ajpheart.physiology.org/cgi/content/abstract/299/4/H1174

online access status

1 reference

Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?resulttype=core&format=json&query=SRC%3AMED%20AND%20EXT_ID%3A20693399

18 June 2024

https://www.physiology.org/doi/pdf/10.1152/ajpheart.00116.2010

1 reference

10.1152/AJPHEART.00116.2010

https://api.crossref.org/works/10.1152/AJPHEART.00116.2010

18 June 2024

American Journal of Physiology Heart and Circulatory Physiology

1 reference

Europe PubMed Central

PubMed publication ID

11 November 2017

299

1 reference

Europe PubMed Central

PubMed publication ID

11 November 2017

4

1 reference

Europe PubMed Central

PubMed publication ID

11 November 2017

H1174-82

1 reference

Europe PubMed Central

PubMed publication ID

11 November 2017

The ceiling effect of pharmacological postconditioning with the phytoestrogen genistein is reversed by the GSK3beta inhibitor SB 216763 [3-(2,4-dichlorophenyl)-4(1-methyl-1H-indol-3-yl)-1H-pyrrole-2,5-dione] through mitochondrial ATP-dependent potas

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00116.2010

21 January 2018

Postconditioning via stuttering reperfusion limits myocardial infarct size in rabbit hearts: role of ERK1/2.

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00116.2010

21 January 2018

Blockade of ATP-sensitive potassium channels prevents myocardial preconditioning in dogs

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00116.2010

21 January 2018

Mitochondrial permeability transition pore opening during myocardial reperfusion--a target for cardioprotection

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00116.2010

21 January 2018

ATP-sensitive K(+) channel activation by nitric oxide and protein kinase G in rabbit ventricular myocytes

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00116.2010

21 January 2018

Inhibiting mitochondrial permeability transition pore opening at reperfusion protects against ischaemia-reperfusion injury

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00116.2010

21 January 2018

Glycogen synthase kinase-3beta mediates convergence of protection signaling to inhibit the mitochondrial permeability transition pore

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00116.2010

21 January 2018

A novel anti-diabetic drug, miglitol, markedly reduces myocardial infarct size in rabbits

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00116.2010

21 January 2018

Acceleration of the healing process and myocardial regeneration may be important as a mechanism of improvement of cardiac function and remodeling by postinfarction granulocyte colony-stimulating factor treatment

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00116.2010

21 January 2018

Effect of myocardial oxygen consumption on infarct size in experimental coronary artery occlusion

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00116.2010

21 January 2018

Mobilized bone marrow cells repair the infarcted heart, improving function and survival

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00116.2010

21 January 2018

Activation of mitochondrial ATP-dependent potassium channels by nitric oxide.

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00116.2010

21 January 2018

Postconditioning: a form of "modified reperfusion" protects the myocardium by activating the phosphatidylinositol 3-kinase-Akt pathway

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00116.2010

21 January 2018

Phosphorylation of the IKs channel complex inhibits drug block: novel mechanism underlying variable antiarrhythmic drug actions

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00116.2010

21 January 2018

Multiple, brief coronary occlusions during early reperfusion protect rabbit hearts by targeting cell signaling pathways.

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00116.2010

21 January 2018

Inhibition of myocardial injury by ischemic postconditioning during reperfusion: comparison with ischemic preconditioning

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00116.2010

21 January 2018

Ischemic postconditioning protects remodeled myocardium via the PI3K-PKB/Akt reperfusion injury salvage kinase pathway.

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00116.2010

21 January 2018

The mechanism by which the mitochondrial ATP-sensitive K+ channel opening and H2O2 inhibit the mitochondrial permeability transition

1

1 reference

10.1152/AJPHEART.00116.2010

https://api.crossref.org/works/10.1152/AJPHEART.00116.2010

18 June 2024

G-CSF prevents cardiac remodeling after myocardial infarction by activating the Jak-Stat pathway in cardiomyocytes

7

1 reference

10.1152/AJPHEART.00116.2010

https://api.crossref.org/works/10.1152/AJPHEART.00116.2010

18 June 2024

Early ischemia after complete coronary ligation in the rabbit, dog, pig, and monkey

8

1 reference

10.1152/AJPHEART.00116.2010

https://api.crossref.org/works/10.1152/AJPHEART.00116.2010

18 June 2024

Intracoronary injection of granulocyte colony-stimulating factor ameliorates the progression of left ventricular remodeling after myocardial ischemia/reperfusion in rabbits

9

1 reference

10.1152/AJPHEART.00116.2010

https://api.crossref.org/works/10.1152/AJPHEART.00116.2010

18 June 2024

Cytokine therapy prevents left ventricular remodeling and dysfunction after myocardial infarction through neovascularization

16

1 reference

10.1152/AJPHEART.00116.2010

https://api.crossref.org/works/10.1152/AJPHEART.00116.2010

18 June 2024

Granulocyte colony stimulating factor directly inhibits myocardial ischemia-reperfusion injury through Akt-endothelial NO synthase pathway

20

1 reference

10.1152/AJPHEART.00116.2010

https://api.crossref.org/works/10.1152/AJPHEART.00116.2010

18 June 2024

Identifiers

10.1152/AJPHEART.00116.2010

1 reference

Europe PubMed Central

PubMed publication ID

11 November 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

11 November 2017

ResearchGate publication ID

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