(Q42943941)

11 November 2017

title

The extinction of morphine-induced conditioned place preference by histone deacetylase inhibition (English)

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11 November 2017

Ru Wang

1

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11 November 2017

Yan Zhang

2

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11 November 2017

Hua Qing

3

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11 November 2017

Mei Liu

4

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11 November 2017

Peng Yang

5

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11 November 2017

language of work or name

English

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publication date

5 August 2010

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full work available at URL

11 November 2017

https://api.elsevier.com/content/article/PII:S0304394010010177?httpAccept=text/xml

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https://api.crossref.org/works/10.1016/J.NEULET.2010.07.080

https://api.elsevier.com/content/article/PII:S0304394010010177?httpAccept=text/plain

18 June 2024

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https://api.crossref.org/works/10.1016/J.NEULET.2010.07.080

18 June 2024

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11 November 2017

volume

483

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11 November 2017

issue

2

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11 November 2017

page(s)

137-142

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Chromatin acetylation, memory, and LTP are impaired in CBP+/- mice: a model for the cognitive deficit in Rubinstein-Taybi syndrome and its amelioration.

11 November 2017

cites work

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Epigenetics: a landscape takes shape

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Integration of long-term-memory-related synaptic plasticity involves bidirectional regulation of gene expression and chromatin structure

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Histone deacetylase inhibitors modulates the induction and expression of amphetamine-induced behavioral sensitization partially through an associated learning of the environment in mice

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Chromatin modifications and their function

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Chromatin remodeling is a key mechanism underlying cocaine-induced plasticity in striatum

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Systemic or intrahippocampal delivery of histone deacetylase inhibitors facilitates fear extinction

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CREB-binding protein controls response to cocaine by acetylating histones at the fosB promoter in the mouse striatum

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Central amygdala extracellular signal-regulated kinase signaling pathway is critical to incubation of opiate craving

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Modulation of chromatin modification facilitates extinction of cocaine-induced conditioned place preference

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Histone deacetylase 5 epigenetically controls behavioral adaptations to chronic emotional stimuli

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Delta FosB mediates epigenetic desensitization of the c-fos gene after chronic amphetamine exposure

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Selective boosting of transcriptional and behavioral responses to drugs of abuse by histone deacetylase inhibition.

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Drug-induced activation of dopamine D(1) receptor signaling and inhibition of class I/II histone deacetylase induce chromatin remodeling in reward circuitry and modulate cocaine-related behaviors

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Short chain fatty acids induce TH gene expression via ERK-dependent phosphorylation of CREB protein.

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Neurotrophic signaling cascades in the pathophysiology and treatment of bipolar disorder

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Modulation of long-term memory for object recognition via HDAC inhibition

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Histone deacetylase inhibitors enhance memory and synaptic plasticity via CREB:CBP-dependent transcriptional activation

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Stress impairs reconsolidation of drug memory via glucocorticoid receptors in the basolateral amygdala.

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Identifiers

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11 November 2017

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